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Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous?
BACKGROUND: Secundum-type atrial septal defects (ASD) constitute 8% to 10% of congenital heart defect. Secundum ASDs can be closed either percutaneously or surgically. However, ASD device closure has proven to be technically safe and feasible; it is not free of complications. These complications inc...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Springer Berlin Heidelberg
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8179864/ https://www.ncbi.nlm.nih.gov/pubmed/34089387 http://dx.doi.org/10.1186/s43044-021-00171-8 |
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author | Khajali, Zahra Firouzi, Ata Pouraliakbar, Hamidreza Hosseini, Zahra Jorfi, Fateme |
author_facet | Khajali, Zahra Firouzi, Ata Pouraliakbar, Hamidreza Hosseini, Zahra Jorfi, Fateme |
author_sort | Khajali, Zahra |
collection | PubMed |
description | BACKGROUND: Secundum-type atrial septal defects (ASD) constitute 8% to 10% of congenital heart defect. Secundum ASDs can be closed either percutaneously or surgically. However, ASD device closure has proven to be technically safe and feasible; it is not free of complications. These complications include device embolization/malposition which have been reported in 3.5% of cases, arrhythmia, and pericardial effusion in 2.6% and 0.5–1.5% respectively, device thrombus, residual shunting, and impingement of the device on the adjacent structures. CASE PRESENTATION: We introduce three patients with secundum ASD who had preexisting pericardial effusion, device closure was performed for them, and after the procedure, the effusion size progressed significantly. We used multimodality imaging to diagnose the cause of pericardial effusion (PE). Cardiac erosion was diagnosed in one of the patients that managed surgically. We did not found any specific procedure-related cause for worsening the pericardial effusion in the other two patients. CONCLUSION: Several reasons include procedure-related complication and other systemic causes should be considered in patients who develop pericardial effusion after trans catheter closure of ASDs. |
format | Online Article Text |
id | pubmed-8179864 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | Springer Berlin Heidelberg |
record_format | MEDLINE/PubMed |
spelling | pubmed-81798642021-06-17 Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? Khajali, Zahra Firouzi, Ata Pouraliakbar, Hamidreza Hosseini, Zahra Jorfi, Fateme Egypt Heart J Case Report BACKGROUND: Secundum-type atrial septal defects (ASD) constitute 8% to 10% of congenital heart defect. Secundum ASDs can be closed either percutaneously or surgically. However, ASD device closure has proven to be technically safe and feasible; it is not free of complications. These complications include device embolization/malposition which have been reported in 3.5% of cases, arrhythmia, and pericardial effusion in 2.6% and 0.5–1.5% respectively, device thrombus, residual shunting, and impingement of the device on the adjacent structures. CASE PRESENTATION: We introduce three patients with secundum ASD who had preexisting pericardial effusion, device closure was performed for them, and after the procedure, the effusion size progressed significantly. We used multimodality imaging to diagnose the cause of pericardial effusion (PE). Cardiac erosion was diagnosed in one of the patients that managed surgically. We did not found any specific procedure-related cause for worsening the pericardial effusion in the other two patients. CONCLUSION: Several reasons include procedure-related complication and other systemic causes should be considered in patients who develop pericardial effusion after trans catheter closure of ASDs. Springer Berlin Heidelberg 2021-06-05 /pmc/articles/PMC8179864/ /pubmed/34089387 http://dx.doi.org/10.1186/s43044-021-00171-8 Text en © The Author(s) 2021 https://creativecommons.org/licenses/by/4.0/Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Case Report Khajali, Zahra Firouzi, Ata Pouraliakbar, Hamidreza Hosseini, Zahra Jorfi, Fateme Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? |
title | Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? |
title_full | Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? |
title_fullStr | Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? |
title_full_unstemmed | Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? |
title_short | Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous? |
title_sort | case report of worsening of preexisting pericardial effusion after asd device closure: is it calamitous? |
topic | Case Report |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8179864/ https://www.ncbi.nlm.nih.gov/pubmed/34089387 http://dx.doi.org/10.1186/s43044-021-00171-8 |
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