Cargando…

The CAGE–MiR-181b-5p–S1PR1 Axis Regulates Anticancer Drug Resistance and Autophagy in Gastric Cancer Cells

Cancer-associated gene (CAGE), a cancer/testis antigen, has been known to promote anticancer drug resistance. Since the underlying mechanisms of CAGE-promoted anticancer drug resistance are poorly understood, we established Anticancer drug-resistant gastric cancer cells (AGS(R)) to better elucidate...

Descripción completa

Detalles Bibliográficos
Autores principales:	Yeon, Minjeong, Kim, Youngmi, Pathak, Deepak, Kwon, Eunju, Kim, Dong Young, Jeong, Myeong Seon, Jung, Hyun Suk, Jeoung, Dooil
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Frontiers Media S.A. 2021
Materias:	Cell and Developmental Biology
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8185229/ https://www.ncbi.nlm.nih.gov/pubmed/34113619 http://dx.doi.org/10.3389/fcell.2021.666387

Ejemplares similares

Histone Deacetylase Inhibitors to Overcome Resistance to Targeted and Immuno Therapy in Metastatic Melanoma
por: Yeon, Minjeong, et al.
Publicado: (2020)

CAGE Binds to Beclin1, Regulates Autophagic Flux and CAGE-Derived Peptide Confers Sensitivity to Anti-cancer Drugs in Non-small Cell Lung Cancer Cells
por: Yeon, Minjeong, et al.
Publicado: (2018)

CAGE-miR-140-5p-Wnt1 Axis Regulates Autophagic Flux, Tumorigenic Potential of Mouse Colon Cancer Cells and Cellular Interactions Mediated by Exosomes
por: Yeon, Minjeong, et al.
Publicado: (2019)

Histone Deacetylase-3/CAGE Axis Targets EGFR Signaling and Regulates the Response to Anti-Cancer Drugs
por: Kim, Hyuna, et al.
Publicado: (2016)

Role of HDAC3-miRNA-CAGE Network in Anti-Cancer Drug-Resistance
por: Kwon, Yoojung, et al.
Publicado: (2018)

MiR-154-5p-MCP1 Axis Regulates Allergic Inflammation by Mediating Cellular Interactions
por: Kim, Misun, et al.
Publicado: (2021)

DDX53 Regulates Cancer Stem Cell-Like Properties by Binding to SOX-2
por: Kim, Youngmi, et al.
Publicado: (2017)

miR-30a Regulates the Expression of CAGE and p53 and Regulates the Response to Anti-Cancer Drugs
por: Park, Deokbum, et al.
Publicado: (2016)

EGR3-HDAC6-IL-27 Axis Mediates Allergic Inflammation and Is Necessary for Tumorigenic Potential of Cancer Cells Enhanced by Allergic Inflammation-Promoted Cellular Interactions
por: Kwon, Yoojung, et al.
Publicado: (2021)

DDX53 Promotes Cancer Stem Cell-Like Properties and Autophagy
por: Kim, Hyuna, et al.
Publicado: (2017)

Targeting Autophagy for Overcoming Resistance to Anti-EGFR Treatments
por: Kwon, Yoojung, et al.
Publicado: (2019)

FcεRI-HDAC3-MCP1 Signaling Axis Promotes Passive Anaphylaxis Mediated by Cellular Interactions
por: Kim, Misun, et al.
Publicado: (2019)

The pentapeptide Gly-Thr-Gly-Lys-Thr confers sensitivity to anti-cancer drugs by inhibition of CAGE binding to GSK3β and decreasing the expression of cyclinD1
por: Kim, Youngmi, et al.
Publicado: (2017)

miR-217 and CAGE form feedback loop and regulates the response to anti-cancer drugs through EGFR and HER2
por: Kim, Youngmi, et al.
Publicado: (2016)

Homoharringtonine Inhibits Allergic Inflammations by Regulating NF-κB-miR-183-5p-BTG1 Axis
por: Kim, Misun, et al.
Publicado: (2020)

Cancer/testis antigen CAGE mediates osimertinib resistance in non-small cell lung cancer cells and predicts poor prognosis in patients with pulmonary adenocarcinoma
por: Yeon, Minjeong, et al.
Publicado: (2023)

Roles of RNA Methylations in Cancer Progression, Autophagy, and Anticancer Drug Resistance
por: Jo, Hyein, et al.
Publicado: (2023)

Nuclear localization signal domain of HDAC3 is necessary and sufficient for the expression regulation of MDR1
por: Park, Hyunmi, et al.
Publicado: (2014)

Tubulin Beta3 Serves as a Target of HDAC3 and Mediates Resistance to Microtubule-Targeting Drugs
por: Kim, Youngmi, et al.
Publicado: (2015)

MiR-135-5p-p62 Axis Regulates Autophagic Flux, Tumorigenic Potential, and Cellular Interactions Mediated by Extracellular Vesicles During Allergic Inflammation
por: Kim, Misun, et al.
Publicado: (2019)

HDAC6 and CXCL13 Mediate Atopic Dermatitis by Regulating Cellular Interactions and Expression Levels of miR-9 and SIRT1
por: Kwon, Yoojung, et al.
Publicado: (2021)

Human Adipose Tissue-Derived Mesenchymal Stem Cells Attenuate Atopic Dermatitis by Regulating the Expression of MIP-2, miR-122a-SOCS1 Axis, and Th1/Th2 Responses
por: Kim, Misun, et al.
Publicado: (2018)

Exosomal MicroRNAs as Mediators of Cellular Interactions Between Cancer Cells and Macrophages
por: Kwon, Yoojung, et al.
Publicado: (2020)

miR-335 Targets SIAH2 and Confers Sensitivity to Anti-Cancer Drugs by Increasing the Expression of HDAC3
por: Kim, Youngmi, et al.
Publicado: (2015)

miR-122-SOCS1-JAK2 axis regulates allergic inflammation and allergic inflammation-promoted cellular interactions
por: Noh, Kyeonga, et al.
Publicado: (2017)

HDAC3 acts as a negative regulator of angiogenesis
por: Park, Deokbum, et al.
Publicado: (2014)

The Hyaluronic Acid–HDAC3–miRNA Network in Allergic Inflammation
por: Kim, Youngmi, et al.
Publicado: (2015)

The Potential of Senescence as a Target for Developing Anticancer Therapy
por: Jo, Hyein, et al.
Publicado: (2023)

Cancer/Testis Antigens as Targets for RNA-Based Anticancer Therapy
por: Shim, Kyeonghee, et al.
Publicado: (2023)

Targeting HDAC6 to Overcome Autophagy-Promoted Anti-Cancer Drug Resistance
por: Jo, Hyein, et al.
Publicado: (2022)

Structural insights into stressosome assembly
por: Kwon, Eunju, et al.
Publicado: (2019)

The metastasis suppressor CD82/KAI1 inhibits fibronectin adhesion-induced epithelial-to-mesenchymal transition in prostate cancer cells by repressing the associated integrin signaling
por: Lee, Jaeseob, et al.
Publicado: (2016)

Anticancer Activity of the PR Domain of Tumor Suppressor RIZ1
por: Sun, Wanpeng, et al.
Publicado: (2011)

HDAC2 as a target for developing anti-cancer drugs
por: Jo, Hyein, et al.
Publicado: (2023)

C1GALT1, Negatively Regulated by miR-181d-5p, Promotes Tumor Progression via Upregulating RAC1 in Lung Adenocarcinoma
por: Dong, Xiaoxia, et al.
Publicado: (2021)

LncRNA ANRIL-mediated miR-181b-5p/S1PR1 axis is involved in the progression of uremic cardiomyopathy through activating T cells
por: Xu, Ying, et al.
Publicado: (2022)

Hypo-Expression of Tuberin Promotes Adenomyosis via the mTOR1-Autophagy Axis
por: Gu, Ni-Hao, et al.
Publicado: (2021)

Prion protein (PrP) gene-knockout cell lines: insight into functions of the PrP
por: Sakudo, Akikazu, et al.
Publicado: (2015)

BRG1 Activates PR65A Transcription to Regulate NO Bioavailability in Vascular Endothelial Cells
por: Chen, Baoyu, et al.
Publicado: (2020)

Circular RNA cMTO1 Promotes PTEN Expression Through Sponging miR-181b-5p in Liver Fibrosis
por: Jin, Hui, et al.
Publicado: (2020)

Cannot write session to /tmp/vufind_sessions/sess_kd6kfddharun1kkvf0fn66fobi