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Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract
Autores principales: | , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8190033/ https://www.ncbi.nlm.nih.gov/pubmed/34108607 http://dx.doi.org/10.1038/s41598-021-91766-3 |
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author | Luong, Thi My Hanh Matsuda, Katsuya Niino, Daisuke Kurohama, Hirokazu Ito, Masahiro Nakashima, Masahiro |
author_facet | Luong, Thi My Hanh Matsuda, Katsuya Niino, Daisuke Kurohama, Hirokazu Ito, Masahiro Nakashima, Masahiro |
author_sort | Luong, Thi My Hanh |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-8190033 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-81900332021-06-10 Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract Luong, Thi My Hanh Matsuda, Katsuya Niino, Daisuke Kurohama, Hirokazu Ito, Masahiro Nakashima, Masahiro Sci Rep Author Correction Nature Publishing Group UK 2021-06-09 /pmc/articles/PMC8190033/ /pubmed/34108607 http://dx.doi.org/10.1038/s41598-021-91766-3 Text en © The Author(s) 2021 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Author Correction Luong, Thi My Hanh Matsuda, Katsuya Niino, Daisuke Kurohama, Hirokazu Ito, Masahiro Nakashima, Masahiro Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract |
title | Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract |
title_full | Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract |
title_fullStr | Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract |
title_full_unstemmed | Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract |
title_short | Author Correction: Significance of abnormal 53BP1 expression as a novel molecular pathologic parameter of follicular-shaped B-cell lymphoid lesions in human digestive tract |
title_sort | author correction: significance of abnormal 53bp1 expression as a novel molecular pathologic parameter of follicular-shaped b-cell lymphoid lesions in human digestive tract |
topic | Author Correction |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8190033/ https://www.ncbi.nlm.nih.gov/pubmed/34108607 http://dx.doi.org/10.1038/s41598-021-91766-3 |
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