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Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome
When clinical presentation is atypical, especially in a non‐Asian population, the finding of recurrent and refractory hypokalemia can serve as a key diagnostic clue for timely diagnoses and management of thyrotoxic periodic paralysis. In suspected cases, complete thyroid laboratory panel should be m...
Autores principales: | , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8218316/ https://www.ncbi.nlm.nih.gov/pubmed/34188930 http://dx.doi.org/10.1002/ccr3.4443 |
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author | Pattan, Vishwanath Chiu, Ken C. Samoa, Raynald |
author_facet | Pattan, Vishwanath Chiu, Ken C. Samoa, Raynald |
author_sort | Pattan, Vishwanath |
collection | PubMed |
description | When clinical presentation is atypical, especially in a non‐Asian population, the finding of recurrent and refractory hypokalemia can serve as a key diagnostic clue for timely diagnoses and management of thyrotoxic periodic paralysis. In suspected cases, complete thyroid laboratory panel should be measured so that T3 toxicosis is not missed. |
format | Online Article Text |
id | pubmed-8218316 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | John Wiley and Sons Inc. |
record_format | MEDLINE/PubMed |
spelling | pubmed-82183162021-06-28 Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome Pattan, Vishwanath Chiu, Ken C. Samoa, Raynald Clin Case Rep Case Reports When clinical presentation is atypical, especially in a non‐Asian population, the finding of recurrent and refractory hypokalemia can serve as a key diagnostic clue for timely diagnoses and management of thyrotoxic periodic paralysis. In suspected cases, complete thyroid laboratory panel should be measured so that T3 toxicosis is not missed. John Wiley and Sons Inc. 2021-06-22 /pmc/articles/PMC8218316/ /pubmed/34188930 http://dx.doi.org/10.1002/ccr3.4443 Text en © 2021 The Authors. Clinical Case Reports published by John Wiley & Sons Ltd. https://creativecommons.org/licenses/by-nc-nd/4.0/This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ (https://creativecommons.org/licenses/by-nc-nd/4.0/) License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made. |
spellingShingle | Case Reports Pattan, Vishwanath Chiu, Ken C. Samoa, Raynald Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome |
title | Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome |
title_full | Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome |
title_fullStr | Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome |
title_full_unstemmed | Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome |
title_short | Recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected Guillain‐Barre syndrome |
title_sort | recurrent, refractory hypokalemia as a diagnostic clue to thyrotoxic periodic paralysis in a patient with acute kidney injury and suspected guillain‐barre syndrome |
topic | Case Reports |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8218316/ https://www.ncbi.nlm.nih.gov/pubmed/34188930 http://dx.doi.org/10.1002/ccr3.4443 |
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