Cargando…

Intra-Abdominal Hypertension-Induced Gastroesophageal Intussusception: A Rare Complication of Transurethral Resection of a Bladder Tumor

Patient: Male, 81-year-old Final Diagnosis: Gastroesophageal intussusception Symptoms: Diffuse tenderness and distension of the abdomen Medication:— Clinical Procedure: — Specialty: Critical Care Medicine OBJECTIVE: Rare disease BACKGROUND: Gastroesophageal intussusception (GEI) generally develops i...

Descripción completa

Detalles Bibliográficos
Autores principales: Sugaya, Akihiko, Shimazui, Takashi, Kitamura, Nobuya, Tokita, Takashi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: International Scientific Literature, Inc. 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8255080/
https://www.ncbi.nlm.nih.gov/pubmed/34176920
http://dx.doi.org/10.12659/AJCR.930426
Descripción
Sumario:Patient: Male, 81-year-old Final Diagnosis: Gastroesophageal intussusception Symptoms: Diffuse tenderness and distension of the abdomen Medication:— Clinical Procedure: — Specialty: Critical Care Medicine OBJECTIVE: Rare disease BACKGROUND: Gastroesophageal intussusception (GEI) generally develops in patients with risk factors. However, intra-abdominal hypertension (IAH) rarely causes sudden GEI in patients without known risk factors. Endoscopic or surgical intervention is generally performed to reduce GEI. However, when GEI is induced by IAH, intra-abdominal pressure (IAP) decompression can contribute to GEI reduction. CASE REPORT: An 81-year-old man who underwent transurethral resection of bladder tumor (TURBT) for hematuria from a bladder tumor located at the left lateral wall had a deteriorated general status and bladder perforation during surgery in February 2020. The perforated portion was coagulated and treated conservatively using a urinary tract catheter. He was admitted to our Intensive Care Unit (ICU) following surgery after undergoing computed tomography (CT). CT revealed free air, ascites, and intra/retroperitoneal edema due to perfusion fluid leakage, and a new GEI was documented. The GEI required reduction; however, since his IAP increased to 21 mmHg, IAH-induced GEI was diagnosed; ascites drainage for IAP decompression was performed. IAP decreased to 12 mmHg after drainage; on subsequent gastrointestinal endoscopy, the GEI had reduced. His condition improved with no recurrence of GEI, and he was discharged from the ICU on day 8. Since cystography findings on day 26 showed no leakage of the bladder, he was discharged from our hospital on day 31. CONCLUSIONS: We report a case of IAH-induced GEI as a complication of perfusion fluid leakage during TURBT. GEI was reduced by IAP decompression by ascites drainage without endoscopic or surgical intervention.