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Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis

We report a case of reversible symptomatic rhabdomyolysis associated with thyrotoxic hypokalemic periodic paralysis. The patient had neither past medical nor family history of either disorder. The presenting neurological symptoms and signs, serum potassium, and creatine kinase levels returned to nor...

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Autores principales: Sathirapanya, Praewchompoo, Suwanno, Komsai, Sathirapanya, Pornchai
Formato: Online Artículo Texto
Lenguaje:English
Publicado: S. Karger AG 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8255658/
https://www.ncbi.nlm.nih.gov/pubmed/34248576
http://dx.doi.org/10.1159/000516771
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author Sathirapanya, Praewchompoo
Suwanno, Komsai
Sathirapanya, Pornchai
author_facet Sathirapanya, Praewchompoo
Suwanno, Komsai
Sathirapanya, Pornchai
author_sort Sathirapanya, Praewchompoo
collection PubMed
description We report a case of reversible symptomatic rhabdomyolysis associated with thyrotoxic hypokalemic periodic paralysis. The patient had neither past medical nor family history of either disorder. The presenting neurological symptoms and signs, serum potassium, and creatine kinase levels returned to normal without specific treatment. Based on previous case reports, we attributed the combination of the disorders to a mutation of the calcium-gated channel (CACN) gene and its related encoded proteins.
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spelling pubmed-82556582021-07-09 Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis Sathirapanya, Praewchompoo Suwanno, Komsai Sathirapanya, Pornchai Case Rep Neurol Single Case − General Neurology We report a case of reversible symptomatic rhabdomyolysis associated with thyrotoxic hypokalemic periodic paralysis. The patient had neither past medical nor family history of either disorder. The presenting neurological symptoms and signs, serum potassium, and creatine kinase levels returned to normal without specific treatment. Based on previous case reports, we attributed the combination of the disorders to a mutation of the calcium-gated channel (CACN) gene and its related encoded proteins. S. Karger AG 2021-06-16 /pmc/articles/PMC8255658/ /pubmed/34248576 http://dx.doi.org/10.1159/000516771 Text en Copyright © 2021 by S. Karger AG, Basel https://creativecommons.org/licenses/by-nc/4.0/This article is licensed under the Creative Commons Attribution-NonCommercial-4.0 International License (CC BY-NC) (http://www.karger.com/Services/OpenAccessLicense). Usage and distribution for commercial purposes requires written permission.
spellingShingle Single Case − General Neurology
Sathirapanya, Praewchompoo
Suwanno, Komsai
Sathirapanya, Pornchai
Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis
title Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis
title_full Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis
title_fullStr Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis
title_full_unstemmed Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis
title_short Reversible Severe Rhabdomyolysis Associated with Thyrotoxic Hypokalemic Periodic Paralysis
title_sort reversible severe rhabdomyolysis associated with thyrotoxic hypokalemic periodic paralysis
topic Single Case − General Neurology
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8255658/
https://www.ncbi.nlm.nih.gov/pubmed/34248576
http://dx.doi.org/10.1159/000516771
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