Cargando…
TRIM66 Overexpression Promotes Glioma Progression and Regulates Glucose Uptake Through cMyc/GLUT3 Signaling
OBJECTIVE: Tripartite motif 66 (TRIM66) is reported to be closely associated with human cancers. However, the roles of TRIM66 in glioma remain unclear. The present study aimed to investigate the clinical significance and biological roles of TRIM66 in human glioma. METHODS: TRIM66 expression in gliom...
Autores principales: | Song, Yuequn, Meng, Lifang, Yu, Jian, Cao, Zhi, Sun, Jizhou, Zhao, Hongyu |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Dove
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8256720/ https://www.ncbi.nlm.nih.gov/pubmed/34234562 http://dx.doi.org/10.2147/CMAR.S293728 |
Ejemplares similares
-
A proteomic study of cMyc improvement of CHO culture
por: Kuystermans, Darrin, et al.
Publicado: (2010) -
Temporal Requirements of cMyc Protein for Reprogramming Mouse Fibroblasts
por: Heffernan, Corey, et al.
Publicado: (2012) -
TRIM28 is overexpressed in glioma and associated with tumor progression
por: Su, Chunhai, et al.
Publicado: (2018) -
Transformation of normal cells by aberrant activation of YAP via cMyc with TEAD
por: Nishimoto, Masazumi, et al.
Publicado: (2019) -
Loss of MYSM1 inhibits the oncogenic activity of cMYC in B cell lymphoma
por: Lin, Yun Hsiao, et al.
Publicado: (2021)