Cargando…
Exposure to Fine Particulate Matter Air Pollution Alters mRNA and miRNA Expression in Bone Marrow-Derived Endothelial Progenitor Cells from Mice
Exposure to fine particulate matter (PM(2.5)) air pollution is associated with quantitative deficits of circulating endothelial progenitor cells (EPCs) in humans. Related exposures of mice to concentrated ambient PM(2.5) (CAP) likewise reduces levels of circulating EPCs and induces defects in their...
Autores principales: | Li, Xiaohong, Haberzettl, Petra, Conklin, Daniel J., Bhatnagar, Aruni, Rouchka, Eric C., Zhang, Mei, O’Toole, Timothy E. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8307414/ https://www.ncbi.nlm.nih.gov/pubmed/34356074 http://dx.doi.org/10.3390/genes12071058 |
Ejemplares similares
-
Exposure to Fine Particulate Air Pollution Causes Vascular Insulin Resistance by Inducing Pulmonary Oxidative Stress
por: Haberzettl, Petra, et al.
Publicado: (2016) -
Exposure to Ambient Air Fine Particulate Matter Prevents VEGF-Induced Mobilization of Endothelial Progenitor Cells from the Bone Marrow
por: Haberzettl, Petra, et al.
Publicado: (2012) -
Carnosine Supplementation Mitigates the Deleterious Effects of Particulate Matter Exposure in Mice
por: Abplanalp, Wesley, et al.
Publicado: (2019) -
Fine particulate matter air pollution and aortic perivascular adipose tissue: Oxidative stress, leptin, and vascular dysfunction
por: Haberzettl, Petra, et al.
Publicado: (2021) -
Acute exposure to air pollution is associated with novel changes in blood levels of endothelin-1 and circulating angiogenic cells in young, healthy adults
por: Finch, Jordan, et al.
Publicado: (2019)