Dipeptidyl peptidase 4 contributes to Alzheimer’s disease–like defects in a mouse model and is increased in sporadic Alzheimer’s disease brains

The amyloid cascade hypothesis, which proposes a prominent role for full-length amyloid β peptides in Alzheimer’s disease, is currently being questioned. In addition to full-length amyloid β peptide, several N-terminally truncated fragments of amyloid β peptide could well contribute to Alzheimer’s d...

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Detalles Bibliográficos
Autores principales: Valverde, Audrey, Dunys, Julie, Lorivel, Thomas, Debayle, Delphine, Gay, Anne-Sophie, Caillava, Céline, Chami, Mounia, Checler, Frédéric
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Society for Biochemistry and Molecular Biology 2021
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8334387/
https://www.ncbi.nlm.nih.gov/pubmed/34265307
http://dx.doi.org/10.1016/j.jbc.2021.100963

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