Cargando…
Polycystin-1 is required for insulin-like growth factor 1-induced cardiomyocyte hypertrophy
Cardiac hypertrophy is the result of responses to various physiological or pathological stimuli. Recently, we showed that polycystin-1 participates in cardiomyocyte hypertrophy elicited by pressure overload and mechanical stress. Interestingly, polycystin-1 knockdown does not affect phenylephrine-in...
Autores principales: | Fernández, Carolina, Torrealba, Natalia, Altamirano, Francisco, Garrido-Moreno, Valeria, Vásquez-Trincado, César, Flores-Vergara, Raúl, López-Crisosto, Camila, Ocaranza, María Paz, Chiong, Mario, Pedrozo, Zully, Lavandero, Sergio |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8372926/ https://www.ncbi.nlm.nih.gov/pubmed/34407099 http://dx.doi.org/10.1371/journal.pone.0255452 |
Ejemplares similares
-
Herpud1 negatively regulates pathological cardiac hypertrophy by inducing IP3 receptor degradation
por: Torrealba, Natalia, et al.
Publicado: (2017) -
Myristate induces mitochondrial fragmentation and cardiomyocyte hypertrophy through mitochondrial E3 ubiquitin ligase MUL1
por: Vásquez-Trincado, César, et al.
Publicado: (2023) -
Nanoparticle-Mediated Angiotensin-(1-9) Drug Delivery for the Treatment of Cardiac Hypertrophy
por: Sepúlveda-Rivas, Sabrina, et al.
Publicado: (2021) -
Polycystin-1 Is a Crucial Regulator of BIN1 Expression and T-Tubule Remodeling Associated with the Development of Dilated Cardiomyopathy
por: Díaz-Vesga, Magda C., et al.
Publicado: (2022) -
Alteration in mitochondrial Ca(2+) uptake disrupts insulin signaling in hypertrophic cardiomyocytes
por: Gutiérrez, Tomás, et al.
Publicado: (2014)