Cargando…
High glucose-induced upregulation of CD36 promotes inflammation stress via NF-κB in H9c2 cells
Cardiac inflammation serves an important role in the progression of diabetic cardiomyopathy. CD36 (cluster of differentiation 36) mediates inflammation stress in a variety of disease states. The present study investigated CD36 expression in high glucose (HG)-induced H9c2 cells, whether CD36 upregula...
Autores principales: | Han, Baosheng, Wang, Jianzhong, Wu, Jiawei, Yan, Fang, Wang, Yaru, Li, Jun |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
D.A. Spandidos
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8430300/ https://www.ncbi.nlm.nih.gov/pubmed/34490487 http://dx.doi.org/10.3892/mmr.2021.12404 |
Ejemplares similares
-
SIRT1 upregulation protects against liver injury induced by a HFD through inhibiting CD36 and the NF-κB pathway in mouse kupffer cells
por: Niu, Bailin, et al.
Publicado: (2018) -
Lymphocytes upregulate CD36 in adipose tissue and liver
por: Couturier, Jacob, et al.
Publicado: (2019) -
Nobiletin Inhibits CD36-Dependent Tumor Angiogenesis, Migration, Invasion, and Sphere Formation Through the Cd36/Stat3/Nf-Κb Signaling Axis
por: Sp, Nipin, et al.
Publicado: (2018) -
Humanized CD36 (hCD36) mouse model supports the preclinical evaluation of therapeutic candidates targeting CD36
por: Xie, Xiulong, et al.
Publicado: (2023) -
hTERT promotes gastric intestinal metaplasia by upregulating CDX2 via NF-κB signaling pathway
por: Chen, Bai-Jun, et al.
Publicado: (2017)