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Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome
Cortical blindness is characterized by loss of vision due to dysfunction of the visual cortices, most commonly secondary to bilateral ischemic infarcts of the occipital lobe. Other causes include surgery such as aortic valve replacement, laryngeal surgery, craniotomy, cerebral angiography, head trau...
Autores principales: | , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Taylor & Francis
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8462920/ https://www.ncbi.nlm.nih.gov/pubmed/34567463 http://dx.doi.org/10.1080/20009666.2021.1974730 |
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author | Shanmugam, S Haver, HL Knecht, SM Rajjoub, R Ali, O Chow, R |
author_facet | Shanmugam, S Haver, HL Knecht, SM Rajjoub, R Ali, O Chow, R |
author_sort | Shanmugam, S |
collection | PubMed |
description | Cortical blindness is characterized by loss of vision due to dysfunction of the visual cortices, most commonly secondary to bilateral ischemic infarcts of the occipital lobe. Other causes include surgery such as aortic valve replacement, laryngeal surgery, craniotomy, cerebral angiography, head trauma, and partial seizures. Visual anosognosia is a distinct feature of cortical blindness, wherein patients claim they can see and confabulate visual perceptions, despite loss of sight. We herewith present a rare phenomenon known as Anton Syndrome, an eponym named after the Austrian neurologist and psychiatrist, Gabriel Anton (1858–1933). There are a limited number of cases of Anton’s Syndrome in the literature, with only 28 case reports published from 1965–2016. Although he was bestowed a neurologic eponym, Anton was an advocate of eugenics and racial hygiene. He publicly advocated for ‘superior breeding’ and ‘selection’ in order to ‘build a brave and noble race.’ We therefore propose replacing the eponym with Bilateral Occipital Lobe Infarct Neglect Deficit (BLIND) Syndrome, with intention of raising awareness of this unique presentation as well as of the widespread interest in eugenics in the early 1900s amongst physicians, notably Gabriel Anton. |
format | Online Article Text |
id | pubmed-8462920 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | Taylor & Francis |
record_format | MEDLINE/PubMed |
spelling | pubmed-84629202021-09-25 Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome Shanmugam, S Haver, HL Knecht, SM Rajjoub, R Ali, O Chow, R J Community Hosp Intern Med Perspect Case Report Cortical blindness is characterized by loss of vision due to dysfunction of the visual cortices, most commonly secondary to bilateral ischemic infarcts of the occipital lobe. Other causes include surgery such as aortic valve replacement, laryngeal surgery, craniotomy, cerebral angiography, head trauma, and partial seizures. Visual anosognosia is a distinct feature of cortical blindness, wherein patients claim they can see and confabulate visual perceptions, despite loss of sight. We herewith present a rare phenomenon known as Anton Syndrome, an eponym named after the Austrian neurologist and psychiatrist, Gabriel Anton (1858–1933). There are a limited number of cases of Anton’s Syndrome in the literature, with only 28 case reports published from 1965–2016. Although he was bestowed a neurologic eponym, Anton was an advocate of eugenics and racial hygiene. He publicly advocated for ‘superior breeding’ and ‘selection’ in order to ‘build a brave and noble race.’ We therefore propose replacing the eponym with Bilateral Occipital Lobe Infarct Neglect Deficit (BLIND) Syndrome, with intention of raising awareness of this unique presentation as well as of the widespread interest in eugenics in the early 1900s amongst physicians, notably Gabriel Anton. Taylor & Francis 2021-09-20 /pmc/articles/PMC8462920/ /pubmed/34567463 http://dx.doi.org/10.1080/20009666.2021.1974730 Text en © 2021 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group on behalf of Greater Baltimore Medical Center. https://creativecommons.org/licenses/by-nc/4.0/This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/ (https://creativecommons.org/licenses/by-nc/4.0/) ), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | Case Report Shanmugam, S Haver, HL Knecht, SM Rajjoub, R Ali, O Chow, R Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome |
title | Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome |
title_full | Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome |
title_fullStr | Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome |
title_full_unstemmed | Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome |
title_short | Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome |
title_sort | bilateral occipital lobe infarct neglect deficit (blind) syndrome |
topic | Case Report |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8462920/ https://www.ncbi.nlm.nih.gov/pubmed/34567463 http://dx.doi.org/10.1080/20009666.2021.1974730 |
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