Cargando…
Herpes simplex virus 1 evades cellular antiviral response by inducing microRNA-24, which attenuates STING synthesis
STING is a nodal point for cellular innate immune response to microbial infections, autoimmunity and cancer; it triggers the synthesis of the antiviral proteins, type I interferons. Many DNA viruses, including Herpes Simplex Virus 1 (HSV1), trigger STING signaling causing inhibition of virus replica...
Autores principales: | Sharma, Nikhil, Wang, Chenyao, Kessler, Patricia, Sen, Ganes C. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8483329/ https://www.ncbi.nlm.nih.gov/pubmed/34591940 http://dx.doi.org/10.1371/journal.ppat.1009950 |
Ejemplares similares
-
STING-Mediated Interferon Induction by Herpes Simplex Virus 1 Requires the Protein Tyrosine Kinase Syk
por: Wang, Chenyao, et al.
Publicado: (2021) -
Herpes simplex virus protein UL56 inhibits cGAS-Mediated DNA sensing to evade antiviral immunity
por: Zheng, Zhou-Qin, et al.
Publicado: (2022) -
Integrity of the Antiviral STING-mediated DNA Sensing in Tumor Cells Is Required to Sustain the Immunotherapeutic Efficacy of Herpes Simplex Oncolytic Virus
por: Froechlich, Guendalina, et al.
Publicado: (2020) -
Effect of combinations of antiviral drugs on herpes simplex encephalitis
por: Gebhardt, Bryan M, et al.
Publicado: (2009) -
Evasion of Early Antiviral Responses by Herpes Simplex Viruses
por: Suazo, Paula A., et al.
Publicado: (2015)