Cargando…
Samm50 Promotes Hypertrophy by Regulating Pink1-Dependent Mitophagy Signaling in Neonatal Cardiomyocytes
Pathological cardiac hypertrophy, the adaptive response of the myocardium to various pathological stimuli, is one of the primary predictors and predisposing factors of heart failure. However, its molecular mechanisms underlying pathogenesis remain poorly understood. Here, we studied the function of...
Autores principales: | Xu, Ran, Kang, Le, Wei, Siang, Yang, Chunjie, Fu, Yuanfeng, Ding, Zhiwen, Zou, Yunzeng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8493082/ https://www.ncbi.nlm.nih.gov/pubmed/34631840 http://dx.doi.org/10.3389/fcvm.2021.748156 |
Ejemplares similares
-
SAMM50 acts with p62 in piecemeal basal- and OXPHOS-induced mitophagy of SAM and MICOS components
por: Abudu, Yakubu Princely, et al.
Publicado: (2021) -
NAP1L5 Promotes Nucleolar Hypertrophy and Is Required for Translation Activation During Cardiomyocyte Hypertrophy
por: Guo, Ningning, et al.
Publicado: (2021) -
Identification of early cardiac dysfunction and heterogeneity after pressure and volume overload in mice by high-frequency echocardiographic strain imaging
por: Xu, Ran, et al.
Publicado: (2023) -
The β(2)-Subunit of Voltage-Gated Calcium Channels Regulates Cardiomyocyte Hypertrophy
por: Pickel, Simone, et al.
Publicado: (2021) -
Informationen der SAMM
Publicado: (2020)