Cargando…
Differential Association of 4E-BP2-Interacting Proteins Is Related to Selective Delayed Neuronal Death after Ischemia
Cerebral ischemia induces an inhibition of protein synthesis and causes cell death and neuronal deficits. These deleterious effects do not occur in resilient areas of the brain, where protein synthesis is restored. In cellular stress conditions, as brain ischemia, translational repressors named euka...
Autores principales: | Martínez-Alonso, Emma, Guerra-Pérez, Natalia, Escobar-Peso, Alejandro, Regidor, Ignacio, Masjuan, Jaime, Alcázar, Alberto |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8509075/ https://www.ncbi.nlm.nih.gov/pubmed/34638676 http://dx.doi.org/10.3390/ijms221910327 |
Ejemplares similares
-
Dihydropyrimidinase-Related Protein 2 Is a New Partner in the Binding between 4E-BP2 and eIF4E Related to Neuronal Death after Cerebral Ischemia
por: Martínez-Alonso, Emma, et al.
Publicado: (2023) -
Phosphorylation of Eukaryotic Initiation Factor 4G1 (eIF4G1) at Ser1147 Is Specific for eIF4G1 Bound to eIF4E in Delayed Neuronal Death after Ischemia
por: Martínez-Alonso, Emma, et al.
Publicado: (2022) -
Dissociation of eIF4E-Binding Protein 2 (4E-BP2) from eIF4E Independent of Thr(37)/Thr(46) Phosphorylation in the Ischemic Stress Response
por: Ayuso, María I., et al.
Publicado: (2015) -
Improving the Efficacy of Quinolylnitrones for Ischemic Stroke Therapy, QN4 and QN15 as New Neuroprotective Agents after Oxygen–Glucose Deprivation/Reoxygenation-Induced Neuronal Injury
por: Alonso, José M., et al.
Publicado: (2022) -
Preclinical Characterization of Antioxidant Quinolyl Nitrone QN23 as a New Candidate for the Treatment of Ischemic Stroke
por: Martínez-Alonso, Emma, et al.
Publicado: (2022)