Secondary hypoxic ischemia alters neurobehavioral outcomes, neuroinflammation, and oxidative stress in mice exposed to controlled cortical impact

OBJECTIVE: Hypoxic ischemia (HI) is a secondary insult that can cause fatal neurologic outcomes after traumatic brain injury (TBI), ranging from mild cognitive deficits to persistent vegetative states. We here aimed to unravel the underlying pathological mechanisms of HI injury in a TBI mouse model....

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Detalles Bibliográficos
Autores principales: Oh, Se-Kwang, Park, Hyun-Jeong, Yu, Gyeong-Gyu, Jeong, Seong-Hae, Lee, Suk-Woo, Kim, Hoon
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Korean Society of Emergency Medicine 2021
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8517469/
https://www.ncbi.nlm.nih.gov/pubmed/34649410
http://dx.doi.org/10.15441/ceem.20.124

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8517469/
https://www.ncbi.nlm.nih.gov/pubmed/34649410
http://dx.doi.org/10.15441/ceem.20.124

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