Cargando…
Regulation of Phosphorylated State of NMDA Receptor by STEP(61) Phosphatase after Mild-Traumatic Brain Injury: Role of Oxidative Stress
Traumatic Brain Injury (TBI) mediates neuronal death through several events involving many molecular pathways, including the glutamate-mediated excitotoxicity for excessive stimulation of N-methyl-D-aspartate receptors (NMDARs), producing activation of death signaling pathways. However, the contribu...
Autores principales: | Carvajal, Francisco J., Cerpa, Waldo |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8533270/ https://www.ncbi.nlm.nih.gov/pubmed/34679709 http://dx.doi.org/10.3390/antiox10101575 |
Ejemplares similares
-
Role of NMDA Receptor-Mediated Glutamatergic Signaling in Chronic and Acute Neuropathologies
por: Carvajal, Francisco J., et al.
Publicado: (2016) -
Exo70 intracellular redistribution after repeated mild traumatic brain injury
por: Lira, Matías, et al.
Publicado: (2021) -
Mild Traumatic Brain Injury Induces Mitochondrial Calcium Overload and Triggers the Upregulation of NCLX in the Hippocampus
por: Mira, Rodrigo G., et al.
Publicado: (2023) -
Regulation of STEP(61) and tyrosine-phosphorylation of NMDA and AMPA receptors during homeostatic synaptic plasticity
por: Jang, Sung-Soo, et al.
Publicado: (2015) -
Traumatic Brain Injury: Mechanisms of Glial Response
por: Mira, Rodrigo G., et al.
Publicado: (2021)