Cargando…
SH3-Binding Glutamic Acid Rich-Deficiency Augments Apoptosis in Neonatal Rat Cardiomyocytes
Congenital heart disease (CHD) is one of the most common birth defects in humans, present in around 40% of newborns with Down’s syndrome (DS). The SH3 domain-binding glutamic acid-rich (SH3BGR) gene, which maps to the DS region, belongs to a gene family encoding a cluster of small thioredoxin-like p...
Autores principales: | Deshpande, Anushka, Borlepawar, Ankush, Rosskopf, Alexandra, Frank, Derk, Frey, Norbert, Rangrez, Ashraf Yusuf |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8541172/ https://www.ncbi.nlm.nih.gov/pubmed/34681711 http://dx.doi.org/10.3390/ijms222011042 |
Ejemplares similares
-
Dysbindin deficiency Alters Cardiac BLOC-1 Complex and Myozap Levels in Mice
por: Borlepawar, Ankush, et al.
Publicado: (2020) -
The E3 ubiquitin ligase HectD3 attenuates cardiac hypertrophy and inflammation in mice
por: Rangrez, Ashraf Yusuf, et al.
Publicado: (2020) -
SRF: a seriously responsible factor in cardiac development and disease
por: Deshpande, Anushka, et al.
Publicado: (2022) -
Dysbindin is a potent inducer of RhoA–SRF-mediated cardiomyocyte hypertrophy
por: Rangrez, Ashraf Yusuf, et al.
Publicado: (2013) -
MicroRNA miR-301a is a novel cardiac regulator of Cofilin-2
por: Rangrez, Ashraf Yusuf, et al.
Publicado: (2017)