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GPI-80 Augments NF-κB Activation in Tumor Cells

Recent studies have discovered a relationship between glycosylphosphatidylinositol (GPI)-anchored protein 80 (GPI-80)/VNN2 (80 kDa GPI-anchored protein) and malignant tumors. GPI-80 is known to regulate neutrophil adhesion; however, the action of GPI-80 on tumors is still obscure. In this study, alt...

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Autores principales: Takeda, Yuji, Kurota, Yuta, Kato, Tomoyuki, Ito, Hiromi, Araki, Akemi, Nara, Hidetoshi, Saitoh, Shinichi, Tanaka, Nobuyuki, Tsuchiya, Norihiko, Asao, Hironobu
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
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Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8584666/
https://www.ncbi.nlm.nih.gov/pubmed/34769456
http://dx.doi.org/10.3390/ijms222112027
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author Takeda, Yuji
Kurota, Yuta
Kato, Tomoyuki
Ito, Hiromi
Araki, Akemi
Nara, Hidetoshi
Saitoh, Shinichi
Tanaka, Nobuyuki
Tsuchiya, Norihiko
Asao, Hironobu
author_facet Takeda, Yuji
Kurota, Yuta
Kato, Tomoyuki
Ito, Hiromi
Araki, Akemi
Nara, Hidetoshi
Saitoh, Shinichi
Tanaka, Nobuyuki
Tsuchiya, Norihiko
Asao, Hironobu
author_sort Takeda, Yuji
collection PubMed
description Recent studies have discovered a relationship between glycosylphosphatidylinositol (GPI)-anchored protein 80 (GPI-80)/VNN2 (80 kDa GPI-anchored protein) and malignant tumors. GPI-80 is known to regulate neutrophil adhesion; however, the action of GPI-80 on tumors is still obscure. In this study, although the expression of GPI-80 mRNA was detectable in several tumor cell lines, the levels of GPI-80 protein were significantly lower than that in neutrophils. To clarify the function of GPI-80 in tumor cells, GPI-80-expressing cells and GPI-80/VNN2 gene-deleted cells were established using PC3 prostate cancer cells. In GPI-80-expressing cells, GPI-80 was mainly detected in vesicles. Furthermore, soluble GPI-80 in the conditioned medium was associated with the exosome marker CD63 and was also detected in the plasma obtained from prostate cancer patients. Unexpectedly, cell adhesion and migration of GPI-80-expressing PC3 cells were not modulated by anti-GPI-80 antibody treatment. However, similar to the GPI-80 family molecule, VNN1, the pantetheinase activity and oxidative state were augmented in GPI-80-expressing cells. GPI-80-expressing cells facilitated non-adhesive proliferation, slow cell proliferation, NF-κB activation and IL-1β production. These phenomena are known to be induced by physiological elevation of the oxidative state. Thus, these observations indicated that GPI-80 affects various tumor responses related to oxidation.
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spelling pubmed-85846662021-11-12 GPI-80 Augments NF-κB Activation in Tumor Cells Takeda, Yuji Kurota, Yuta Kato, Tomoyuki Ito, Hiromi Araki, Akemi Nara, Hidetoshi Saitoh, Shinichi Tanaka, Nobuyuki Tsuchiya, Norihiko Asao, Hironobu Int J Mol Sci Article Recent studies have discovered a relationship between glycosylphosphatidylinositol (GPI)-anchored protein 80 (GPI-80)/VNN2 (80 kDa GPI-anchored protein) and malignant tumors. GPI-80 is known to regulate neutrophil adhesion; however, the action of GPI-80 on tumors is still obscure. In this study, although the expression of GPI-80 mRNA was detectable in several tumor cell lines, the levels of GPI-80 protein were significantly lower than that in neutrophils. To clarify the function of GPI-80 in tumor cells, GPI-80-expressing cells and GPI-80/VNN2 gene-deleted cells were established using PC3 prostate cancer cells. In GPI-80-expressing cells, GPI-80 was mainly detected in vesicles. Furthermore, soluble GPI-80 in the conditioned medium was associated with the exosome marker CD63 and was also detected in the plasma obtained from prostate cancer patients. Unexpectedly, cell adhesion and migration of GPI-80-expressing PC3 cells were not modulated by anti-GPI-80 antibody treatment. However, similar to the GPI-80 family molecule, VNN1, the pantetheinase activity and oxidative state were augmented in GPI-80-expressing cells. GPI-80-expressing cells facilitated non-adhesive proliferation, slow cell proliferation, NF-κB activation and IL-1β production. These phenomena are known to be induced by physiological elevation of the oxidative state. Thus, these observations indicated that GPI-80 affects various tumor responses related to oxidation. MDPI 2021-11-06 /pmc/articles/PMC8584666/ /pubmed/34769456 http://dx.doi.org/10.3390/ijms222112027 Text en © 2021 by the authors. https://creativecommons.org/licenses/by/4.0/Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).
spellingShingle Article
Takeda, Yuji
Kurota, Yuta
Kato, Tomoyuki
Ito, Hiromi
Araki, Akemi
Nara, Hidetoshi
Saitoh, Shinichi
Tanaka, Nobuyuki
Tsuchiya, Norihiko
Asao, Hironobu
GPI-80 Augments NF-κB Activation in Tumor Cells
title GPI-80 Augments NF-κB Activation in Tumor Cells
title_full GPI-80 Augments NF-κB Activation in Tumor Cells
title_fullStr GPI-80 Augments NF-κB Activation in Tumor Cells
title_full_unstemmed GPI-80 Augments NF-κB Activation in Tumor Cells
title_short GPI-80 Augments NF-κB Activation in Tumor Cells
title_sort gpi-80 augments nf-κb activation in tumor cells
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8584666/
https://www.ncbi.nlm.nih.gov/pubmed/34769456
http://dx.doi.org/10.3390/ijms222112027
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