Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

Members of the fetal-gene-program may act as regulatory components to impede deleterious events occurring with cardiac remodeling, and constitute potential novel therapeutic heart failure (HF) targets. Mitochondrial energy derangements occur both during early fetal development and in patients with H...

Descripción completa

Detalles Bibliográficos
Autores principales: Bomer, Nils, Pavez-Giani, Mario G., Deiman, Frederik E., Linders, Annet N., Hoes, Martijn F., Baierl, Christiane L.J., Oberdorf-Maass, Silke U., de Boer, Rudolf A., Silljé, Herman H.W., Berezikov, Eugene, Simonides, Warner S., Westenbrink, B. Daan, van der Meer, Peter
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8584701/
https://www.ncbi.nlm.nih.gov/pubmed/34769334
http://dx.doi.org/10.3390/ijms222111906

Ejemplares similares

Ejemplares similares