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Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells

Detalles Bibliográficos
Autores principales: Li, Weidong, Dong, Xuesong, He, Changjun, Tan, Gang, Li, Ziyi, Zhai, Bo, Feng, Jing, Jiang, Xian, Liu, Chang, Jiang, Hongchi, Sun, Xueying
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8638182/
https://www.ncbi.nlm.nih.gov/pubmed/34852817
http://dx.doi.org/10.1186/s13046-021-02183-3
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author Li, Weidong
Dong, Xuesong
He, Changjun
Tan, Gang
Li, Ziyi
Zhai, Bo
Feng, Jing
Jiang, Xian
Liu, Chang
Jiang, Hongchi
Sun, Xueying
author_facet Li, Weidong
Dong, Xuesong
He, Changjun
Tan, Gang
Li, Ziyi
Zhai, Bo
Feng, Jing
Jiang, Xian
Liu, Chang
Jiang, Hongchi
Sun, Xueying
author_sort Li, Weidong
collection PubMed
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spelling pubmed-86381822021-12-02 Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells Li, Weidong Dong, Xuesong He, Changjun Tan, Gang Li, Ziyi Zhai, Bo Feng, Jing Jiang, Xian Liu, Chang Jiang, Hongchi Sun, Xueying J Exp Clin Cancer Res Correction BioMed Central 2021-12-01 /pmc/articles/PMC8638182/ /pubmed/34852817 http://dx.doi.org/10.1186/s13046-021-02183-3 Text en © The Author(s) 2021 https://creativecommons.org/licenses/by/4.0/Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/ (https://creativecommons.org/publicdomain/zero/1.0/) ) applies to the data made available in this article, unless otherwise stated in a credit line to the data.
spellingShingle Correction
Li, Weidong
Dong, Xuesong
He, Changjun
Tan, Gang
Li, Ziyi
Zhai, Bo
Feng, Jing
Jiang, Xian
Liu, Chang
Jiang, Hongchi
Sun, Xueying
Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells
title Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells
title_full Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells
title_fullStr Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells
title_full_unstemmed Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells
title_short Correction to: LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells
title_sort correction to: lncrna snhg1 contributes to sorafenib resistance by activating the akt pathway and is positively regulated by mir-21 in hepatocellular carcinoma cells
topic Correction
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8638182/
https://www.ncbi.nlm.nih.gov/pubmed/34852817
http://dx.doi.org/10.1186/s13046-021-02183-3
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