The Cross Talk Between p53 and mTOR Pathways in Response to Physiological and Genotoxic Stresses

The tumor suppressor p53 is activated upon multiple cellular stresses, including DNA damage, oncogene activation, ribosomal stress, and hypoxia, to induce cell cycle arrest, apoptosis, and senescence. Mammalian target of rapamycin (mTOR), an evolutionarily conserved serine/threonine protein kinase,...

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Detalles Bibliográficos
Autores principales: Cui, Danrui, Qu, Ruirui, Liu, Dian, Xiong, Xiufang, Liang, Tingbo, Zhao, Yongchao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2021
Materias:
Cell and Developmental Biology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8638743/
https://www.ncbi.nlm.nih.gov/pubmed/34869377
http://dx.doi.org/10.3389/fcell.2021.775507

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8638743/
https://www.ncbi.nlm.nih.gov/pubmed/34869377
http://dx.doi.org/10.3389/fcell.2021.775507

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