Cargando…
Etifoxine Restores Mitochondrial Oxidative Phosphorylation and Improves Cognitive Recovery Following Traumatic Brain Injury
The opening of the mitochondrial permeability transition pore (mPTP) has emerged as a pivotal event following traumatic brain injury (TBI). Evidence showing the impact of the translocator protein (TSPO) over mPTP activity has prompted several studies exploring the effect of TSPO ligands, including e...
Autores principales: | Palzur, Eilam, Edelman, Doron, Sakas, Reem, Soustiel, Jean Francois |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8657969/ https://www.ncbi.nlm.nih.gov/pubmed/34884686 http://dx.doi.org/10.3390/ijms222312881 |
Ejemplares similares
-
Reduction of Traumatic Brain Damage by Tspo Ligand Etifoxine
por: Shehadeh, Mona, et al.
Publicado: (2019) -
HBO treatment enhances motor function and modulates pain development after sciatic nerve injury via protection the mitochondrial function
por: Awad-Igbaria, Yaseen, et al.
Publicado: (2023) -
Etifoxine for Pain Patients with Anxiety
por: Choi, Yun Mi, et al.
Publicado: (2015) -
Translocator Protein Ligand Etifoxine Attenuates MPTP-Induced Neurotoxicity
por: Tian, Qi, et al.
Publicado: (2022) -
Etifoxine improves sensorimotor deficits and reduces glial activation, neuronal degeneration, and neuroinflammation in a rat model of traumatic brain injury
por: Simon-O’Brien, Emmanuelle, et al.
Publicado: (2016)