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Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers
Smoking is the most important factor affecting the oral cavity by components born in the tobacco combustion process and acting directly on the oral mucous membranes, dental arch and indirectly on the teeth support. Recent studies show the tobacco action on the oral cavity, manifestations in the form...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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Medical University Publishing House Craiova
2021
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8679161/ https://www.ncbi.nlm.nih.gov/pubmed/35003770 http://dx.doi.org/10.12865/CHSJ.47.03.08 |
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author | POPA, GABRIEL VALERIU MOGOANTĂ, LAURENŢIU BADEA, OANA |
author_facet | POPA, GABRIEL VALERIU MOGOANTĂ, LAURENŢIU BADEA, OANA |
author_sort | POPA, GABRIEL VALERIU |
collection | PubMed |
description | Smoking is the most important factor affecting the oral cavity by components born in the tobacco combustion process and acting directly on the oral mucous membranes, dental arch and indirectly on the teeth support. Recent studies show the tobacco action on the oral cavity, manifestations in the form of gingivitis, bacterial plaque, dental plaque, papillary bleeding at drilling, periodontitis. Purpose of the study. In this study, we have set out to assess the macroscopic modifications of oral cavity on smokers. Materials and Methods. The participants in the study were divided into two groups, the first group of smokers with a smoking period over 5 years and the control group of nonsmokers. The patients in the two groups underwent a physical examination and an objective clinical examination, the resulting data being compared with the control group. Results. For the bacterial plaque indicatorin the smoker group there was obtained a mean value of 35.68±12.45, compared to a mean value of 16.32±6.61 for the nonsmoker group, the dental plaque indicatorfor the smoker group had a mean value of 2.24±1.02, higher than the one in the nonsmoker group, namely 0.94±0.68, and for the drilling bleeding indicator we obtained a mean value of 19.54±7.89 in the nonsmoker group, which is lower than that in the smoker group, namely 42.86±14.93. Conclusions. Smoking is a cause that maintains and aggravates the periodontal disease, including the risk of periodontitis, allowing the aggravation of gingivitis, considered a reversible surface inflammation of the gum mucosa which, by accumulation of dental plaque, the dental plaque accompanied by incorrect oral hygiene, favors the progression to periodontitis. |
format | Online Article Text |
id | pubmed-8679161 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | Medical University Publishing House Craiova |
record_format | MEDLINE/PubMed |
spelling | pubmed-86791612022-01-06 Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers POPA, GABRIEL VALERIU MOGOANTĂ, LAURENŢIU BADEA, OANA Curr Health Sci J Original Paper Smoking is the most important factor affecting the oral cavity by components born in the tobacco combustion process and acting directly on the oral mucous membranes, dental arch and indirectly on the teeth support. Recent studies show the tobacco action on the oral cavity, manifestations in the form of gingivitis, bacterial plaque, dental plaque, papillary bleeding at drilling, periodontitis. Purpose of the study. In this study, we have set out to assess the macroscopic modifications of oral cavity on smokers. Materials and Methods. The participants in the study were divided into two groups, the first group of smokers with a smoking period over 5 years and the control group of nonsmokers. The patients in the two groups underwent a physical examination and an objective clinical examination, the resulting data being compared with the control group. Results. For the bacterial plaque indicatorin the smoker group there was obtained a mean value of 35.68±12.45, compared to a mean value of 16.32±6.61 for the nonsmoker group, the dental plaque indicatorfor the smoker group had a mean value of 2.24±1.02, higher than the one in the nonsmoker group, namely 0.94±0.68, and for the drilling bleeding indicator we obtained a mean value of 19.54±7.89 in the nonsmoker group, which is lower than that in the smoker group, namely 42.86±14.93. Conclusions. Smoking is a cause that maintains and aggravates the periodontal disease, including the risk of periodontitis, allowing the aggravation of gingivitis, considered a reversible surface inflammation of the gum mucosa which, by accumulation of dental plaque, the dental plaque accompanied by incorrect oral hygiene, favors the progression to periodontitis. Medical University Publishing House Craiova 2021 2021-09-30 /pmc/articles/PMC8679161/ /pubmed/35003770 http://dx.doi.org/10.12865/CHSJ.47.03.08 Text en Copyright © 2014, Medical University Publishing House Craiova https://creativecommons.org/licenses/by-nc-sa/4.0/This is an open-access article distributed under the terms of a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International Public License, which permits unrestricted use, adaptation, distribution and reproduction in any medium, non-commercially, provided the new creations are licensed under identical terms as the original work and the original work is properly cited. |
spellingShingle | Original Paper POPA, GABRIEL VALERIU MOGOANTĂ, LAURENŢIU BADEA, OANA Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers |
title | Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers |
title_full | Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers |
title_fullStr | Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers |
title_full_unstemmed | Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers |
title_short | Macroscopic Changes in Oral Mucosa and Hygiene Indicators in Smokers |
title_sort | macroscopic changes in oral mucosa and hygiene indicators in smokers |
topic | Original Paper |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8679161/ https://www.ncbi.nlm.nih.gov/pubmed/35003770 http://dx.doi.org/10.12865/CHSJ.47.03.08 |
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