Cargando…
MUT-7 Provides Molecular Insight into the Werner Syndrome Exonuclease
Werner syndrome (WS) is a rare recessive genetic disease characterized by premature aging. Individuals with this disorder develop normally during childhood, but their physiological conditions exacerbate the aging process in late adolescence. WS is caused by mutation of the human WS gene (WRN), which...
Autores principales: | Hsu, Tsung-Yuan, Hsu, Ling-Nung, Chen, Shih-Yu, Juang, Bi-Tzen |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8700014/ https://www.ncbi.nlm.nih.gov/pubmed/34943966 http://dx.doi.org/10.3390/cells10123457 |
Ejemplares similares
-
C. elegans orthologs MUT-7/CeWRN-1 of Werner syndrome protein regulate neuronal plasticity
por: Hsu, Tsung-Yuan, et al.
Publicado: (2021) -
Sequence-specific processing of telomeric 3' overhangs by the Werner syndrome protein
exonuclease activity
por: Li, Baomin, et al.
Publicado: (2009) -
The Werner Syndrome Helicase/Exonuclease Processes Mobile D-Loops through Branch Migration and Degradation
por: Opresko, Patricia L., et al.
Publicado: (2009) -
MutL sliding clamps coordinate exonuclease-independent Escherichia coli mismatch repair
por: Liu, Jiaquan, et al.
Publicado: (2019) -
A conserved and species-specific functional interaction between the Werner syndrome-like exonuclease atWEX and the Ku heterodimer in Arabidopsis
por: Li, Baomin, et al.
Publicado: (2005)