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Cardiac Metabolism in Sepsis

The mechanism of sepsis-induced cardiac dysfunction is believed to be different from that of myocardial ischemia. In sepsis, chemical mediators, such as endotoxins, cytokines, and nitric oxide, cause metabolic abnormalities, mitochondrial dysfunction, and downregulation of β-adrenergic receptors. Th...

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Detalles Bibliográficos
Autores principales: Kawaguchi, Satoshi, Okada, Motoi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8707959/
https://www.ncbi.nlm.nih.gov/pubmed/34940604
http://dx.doi.org/10.3390/metabo11120846
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author Kawaguchi, Satoshi
Okada, Motoi
author_facet Kawaguchi, Satoshi
Okada, Motoi
author_sort Kawaguchi, Satoshi
collection PubMed
description The mechanism of sepsis-induced cardiac dysfunction is believed to be different from that of myocardial ischemia. In sepsis, chemical mediators, such as endotoxins, cytokines, and nitric oxide, cause metabolic abnormalities, mitochondrial dysfunction, and downregulation of β-adrenergic receptors. These factors inhibit the production of ATP, essential for myocardial energy metabolism, resulting in cardiac dysfunction. This review focuses on the metabolic changes in sepsis, particularly in the heart. In addition to managing inflammation, interventions focusing on metabolism may be a new therapeutic strategy for cardiac dysfunction due to sepsis.
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spelling pubmed-87079592021-12-25 Cardiac Metabolism in Sepsis Kawaguchi, Satoshi Okada, Motoi Metabolites Review The mechanism of sepsis-induced cardiac dysfunction is believed to be different from that of myocardial ischemia. In sepsis, chemical mediators, such as endotoxins, cytokines, and nitric oxide, cause metabolic abnormalities, mitochondrial dysfunction, and downregulation of β-adrenergic receptors. These factors inhibit the production of ATP, essential for myocardial energy metabolism, resulting in cardiac dysfunction. This review focuses on the metabolic changes in sepsis, particularly in the heart. In addition to managing inflammation, interventions focusing on metabolism may be a new therapeutic strategy for cardiac dysfunction due to sepsis. MDPI 2021-12-06 /pmc/articles/PMC8707959/ /pubmed/34940604 http://dx.doi.org/10.3390/metabo11120846 Text en © 2021 by the authors. https://creativecommons.org/licenses/by/4.0/Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).
spellingShingle Review
Kawaguchi, Satoshi
Okada, Motoi
Cardiac Metabolism in Sepsis
title Cardiac Metabolism in Sepsis
title_full Cardiac Metabolism in Sepsis
title_fullStr Cardiac Metabolism in Sepsis
title_full_unstemmed Cardiac Metabolism in Sepsis
title_short Cardiac Metabolism in Sepsis
title_sort cardiac metabolism in sepsis
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8707959/
https://www.ncbi.nlm.nih.gov/pubmed/34940604
http://dx.doi.org/10.3390/metabo11120846
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