LDLR dysfunction induces LDL accumulation and promotes pulmonary fibrosis

Treatments for pulmonary fibrosis (PF) are ineffective because its molecular pathogenesis and therapeutic targets are unclear. Here, we show that the expression of low‐density lipoprotein receptor (LDLR) was significantly decreased in alveolar type II (ATII) and fibroblast cells, whereas it was incr...

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Detalles Bibliográficos
Autores principales: Shi, Xiangguang, Chen, Yahui, Liu, Qingmei, Mei, Xueqian, Liu, Jing, Tang, Yulong, Luo, Ruoyu, Sun, Dayan, Ma, Yanyun, Wu, Wenyu, Tu, Wenzhen, Zhao, Yinhuan, Xu, Weihong, Ke, Yuehai, Jiang, Shuai, Huang, Yan, Zhang, Rui, Wang, Lei, Chen, Yuanyuan, Xia, Jingjing, Pu, Weilin, Zhu, Honglin, Zuo, Xiaoxia, Li, Yisha, Xu, Jinhua, Gao, Fei, Wei, Dong, Chen, Jingyu, Yin, Wenguang, Wang, Qingwen, Dai, Huaping, Yang, Libing, Guo, Gang, Cui, Jimin, Song, Nana, Zou, Hejian, Zhao, Shimin, Distler, Jörg H.W., Jin, Li, Wang, Jiucun
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2022
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8792399/
https://www.ncbi.nlm.nih.gov/pubmed/35083881
http://dx.doi.org/10.1002/ctm2.711

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8792399/
https://www.ncbi.nlm.nih.gov/pubmed/35083881
http://dx.doi.org/10.1002/ctm2.711

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