Cargando…
Synaptic memory requires CaMKII
Long-term potentiation (LTP) is arguably the most compelling cellular model for learning and memory. While the mechanisms underlying the induction of LTP (‘learning’) are well understood, the maintenance of LTP (‘memory’) has remained contentious over the last 20 years. Here, we find that Ca(2+)-cal...
Autores principales: | Tao, Wucheng, Lee, Joel, Chen, Xiumin, Díaz-Alonso, Javier, Zhou, Jing, Pleasure, Samuel, Nicoll, Roger A |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
eLife Sciences Publications, Ltd
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8798046/ https://www.ncbi.nlm.nih.gov/pubmed/34908526 http://dx.doi.org/10.7554/eLife.60360 |
Ejemplares similares
-
Synaptic memory and CaMKII
por: Nicoll, Roger A., et al.
Publicado: (2023) -
CASK and CaMKII function in Drosophila memory
por: Malik, Bilal R., et al.
Publicado: (2014) -
CaMKII Isoforms in Learning and Memory: Localization and Function
por: Zalcman, Gisela, et al.
Publicado: (2018) -
The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms
por: Incontro, Salvatore, et al.
Publicado: (2018) -
Locally-Induced CaMKII Translocation Requires Nucleotide Binding
por: Fitzgerald, Zachary T., et al.
Publicado: (2020)