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Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage
Autores principales: | , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8805271/ https://www.ncbi.nlm.nih.gov/pubmed/35101021 http://dx.doi.org/10.1186/s12915-022-01237-y |
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author | Pandey, Satyaprakash Hajikazemi, Mona Zacheja, Theresa Schalbetter, Stephanie Neale, Matthew J. Baxter, Jonathan Guryev, Victor Hofmann, Andreas Heermann, Dieter W. Juranek, Stefan A. Paeschke, Katrin |
author_facet | Pandey, Satyaprakash Hajikazemi, Mona Zacheja, Theresa Schalbetter, Stephanie Neale, Matthew J. Baxter, Jonathan Guryev, Victor Hofmann, Andreas Heermann, Dieter W. Juranek, Stefan A. Paeschke, Katrin |
author_sort | Pandey, Satyaprakash |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-8805271 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-88052712022-02-03 Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage Pandey, Satyaprakash Hajikazemi, Mona Zacheja, Theresa Schalbetter, Stephanie Neale, Matthew J. Baxter, Jonathan Guryev, Victor Hofmann, Andreas Heermann, Dieter W. Juranek, Stefan A. Paeschke, Katrin BMC Biol Correction BioMed Central 2022-02-01 /pmc/articles/PMC8805271/ /pubmed/35101021 http://dx.doi.org/10.1186/s12915-022-01237-y Text en © The Author(s) 2022 https://creativecommons.org/licenses/by/4.0/Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/ (https://creativecommons.org/publicdomain/zero/1.0/) ) applies to the data made available in this article, unless otherwise stated in a credit line to the data. |
spellingShingle | Correction Pandey, Satyaprakash Hajikazemi, Mona Zacheja, Theresa Schalbetter, Stephanie Neale, Matthew J. Baxter, Jonathan Guryev, Victor Hofmann, Andreas Heermann, Dieter W. Juranek, Stefan A. Paeschke, Katrin Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage |
title | Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage |
title_full | Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage |
title_fullStr | Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage |
title_full_unstemmed | Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage |
title_short | Author Correction to: Telomerase subunit Est2 marks internal sites that are prone to accumulate DNA damage |
title_sort | author correction to: telomerase subunit est2 marks internal sites that are prone to accumulate dna damage |
topic | Correction |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8805271/ https://www.ncbi.nlm.nih.gov/pubmed/35101021 http://dx.doi.org/10.1186/s12915-022-01237-y |
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