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Hormesis and Endothelial Progenitor Cells
Hormetic-biphasic dose response relationships are reported herein for human endothelial progenitor cells involving estradiol, nicotine, the anti-diabetic agent pioglitazone, resveratrol, and progesterone. In general, these studies demonstrate the capacity of these agents to enhance EPC proliferation...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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SAGE Publications
2022
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8874175/ https://www.ncbi.nlm.nih.gov/pubmed/35221821 http://dx.doi.org/10.1177/15593258211068625 |
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author | Calabrese, Edward J. |
author_facet | Calabrese, Edward J. |
author_sort | Calabrese, Edward J. |
collection | PubMed |
description | Hormetic-biphasic dose response relationships are reported herein for human endothelial progenitor cells involving estradiol, nicotine, the anti-diabetic agent pioglitazone, resveratrol, and progesterone. In general, these studies demonstrate the capacity of these agents to enhance EPC proliferation and angiogenesis functional applications, having a focus on repairing endothelial tissue damage due to acute injury (e.g., stroke), as well as damage from chronic conditions (e.g., atherosclerosis) and normal aging processes. |
format | Online Article Text |
id | pubmed-8874175 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | SAGE Publications |
record_format | MEDLINE/PubMed |
spelling | pubmed-88741752022-02-26 Hormesis and Endothelial Progenitor Cells Calabrese, Edward J. Dose Response Review Hormetic-biphasic dose response relationships are reported herein for human endothelial progenitor cells involving estradiol, nicotine, the anti-diabetic agent pioglitazone, resveratrol, and progesterone. In general, these studies demonstrate the capacity of these agents to enhance EPC proliferation and angiogenesis functional applications, having a focus on repairing endothelial tissue damage due to acute injury (e.g., stroke), as well as damage from chronic conditions (e.g., atherosclerosis) and normal aging processes. SAGE Publications 2022-02-23 /pmc/articles/PMC8874175/ /pubmed/35221821 http://dx.doi.org/10.1177/15593258211068625 Text en © The Author(s) 2022 https://creativecommons.org/licenses/by-nc/4.0/This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage). |
spellingShingle | Review Calabrese, Edward J. Hormesis and Endothelial Progenitor Cells |
title | Hormesis and Endothelial Progenitor Cells |
title_full | Hormesis and Endothelial Progenitor Cells |
title_fullStr | Hormesis and Endothelial Progenitor Cells |
title_full_unstemmed | Hormesis and Endothelial Progenitor Cells |
title_short | Hormesis and Endothelial Progenitor Cells |
title_sort | hormesis and endothelial progenitor cells |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8874175/ https://www.ncbi.nlm.nih.gov/pubmed/35221821 http://dx.doi.org/10.1177/15593258211068625 |
work_keys_str_mv | AT calabreseedwardj hormesisandendothelialprogenitorcells |