Cargando…

Hormesis and Endothelial Progenitor Cells

Hormetic-biphasic dose response relationships are reported herein for human endothelial progenitor cells involving estradiol, nicotine, the anti-diabetic agent pioglitazone, resveratrol, and progesterone. In general, these studies demonstrate the capacity of these agents to enhance EPC proliferation...

Descripción completa

Detalles Bibliográficos
Autor principal: Calabrese, Edward J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: SAGE Publications 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8874175/
https://www.ncbi.nlm.nih.gov/pubmed/35221821
http://dx.doi.org/10.1177/15593258211068625
_version_ 1784657625220120576
author Calabrese, Edward J.
author_facet Calabrese, Edward J.
author_sort Calabrese, Edward J.
collection PubMed
description Hormetic-biphasic dose response relationships are reported herein for human endothelial progenitor cells involving estradiol, nicotine, the anti-diabetic agent pioglitazone, resveratrol, and progesterone. In general, these studies demonstrate the capacity of these agents to enhance EPC proliferation and angiogenesis functional applications, having a focus on repairing endothelial tissue damage due to acute injury (e.g., stroke), as well as damage from chronic conditions (e.g., atherosclerosis) and normal aging processes.
format Online
Article
Text
id pubmed-8874175
institution National Center for Biotechnology Information
language English
publishDate 2022
publisher SAGE Publications
record_format MEDLINE/PubMed
spelling pubmed-88741752022-02-26 Hormesis and Endothelial Progenitor Cells Calabrese, Edward J. Dose Response Review Hormetic-biphasic dose response relationships are reported herein for human endothelial progenitor cells involving estradiol, nicotine, the anti-diabetic agent pioglitazone, resveratrol, and progesterone. In general, these studies demonstrate the capacity of these agents to enhance EPC proliferation and angiogenesis functional applications, having a focus on repairing endothelial tissue damage due to acute injury (e.g., stroke), as well as damage from chronic conditions (e.g., atherosclerosis) and normal aging processes. SAGE Publications 2022-02-23 /pmc/articles/PMC8874175/ /pubmed/35221821 http://dx.doi.org/10.1177/15593258211068625 Text en © The Author(s) 2022 https://creativecommons.org/licenses/by-nc/4.0/This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).
spellingShingle Review
Calabrese, Edward J.
Hormesis and Endothelial Progenitor Cells
title Hormesis and Endothelial Progenitor Cells
title_full Hormesis and Endothelial Progenitor Cells
title_fullStr Hormesis and Endothelial Progenitor Cells
title_full_unstemmed Hormesis and Endothelial Progenitor Cells
title_short Hormesis and Endothelial Progenitor Cells
title_sort hormesis and endothelial progenitor cells
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8874175/
https://www.ncbi.nlm.nih.gov/pubmed/35221821
http://dx.doi.org/10.1177/15593258211068625
work_keys_str_mv AT calabreseedwardj hormesisandendothelialprogenitorcells