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Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway
Autores principales: | , , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8891337/ https://www.ncbi.nlm.nih.gov/pubmed/35236822 http://dx.doi.org/10.1038/s41419-022-04631-0 |
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author | Xu, Dafeng Wang, Yu Wu, Jincai Zhang, Zhensheng Chen, Jiacheng Xie, Mingwei Tang, Rong Chen, Cheng Chen, Liang Lin, Shixun Luo, Xiangxiang Zheng, Jinfang |
author_facet | Xu, Dafeng Wang, Yu Wu, Jincai Zhang, Zhensheng Chen, Jiacheng Xie, Mingwei Tang, Rong Chen, Cheng Chen, Liang Lin, Shixun Luo, Xiangxiang Zheng, Jinfang |
author_sort | Xu, Dafeng |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-8891337 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-88913372022-03-08 Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway Xu, Dafeng Wang, Yu Wu, Jincai Zhang, Zhensheng Chen, Jiacheng Xie, Mingwei Tang, Rong Chen, Cheng Chen, Liang Lin, Shixun Luo, Xiangxiang Zheng, Jinfang Cell Death Dis Correction Nature Publishing Group UK 2022-03-02 /pmc/articles/PMC8891337/ /pubmed/35236822 http://dx.doi.org/10.1038/s41419-022-04631-0 Text en © The Author(s) 2022 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Correction Xu, Dafeng Wang, Yu Wu, Jincai Zhang, Zhensheng Chen, Jiacheng Xie, Mingwei Tang, Rong Chen, Cheng Chen, Liang Lin, Shixun Luo, Xiangxiang Zheng, Jinfang Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway |
title | Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway |
title_full | Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway |
title_fullStr | Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway |
title_full_unstemmed | Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway |
title_short | Correction to: ECT2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ECT2/PLK1/PTEN pathway |
title_sort | correction to: ect2 overexpression promotes the polarization of tumor-associated macrophages in hepatocellular carcinoma via the ect2/plk1/pten pathway |
topic | Correction |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8891337/ https://www.ncbi.nlm.nih.gov/pubmed/35236822 http://dx.doi.org/10.1038/s41419-022-04631-0 |
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