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Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness

Mechanical stress is known to fuel several hallmarks of cancer, ranging from genome instability to uncontrolled proliferation or invasion. Cancer cells are constantly challenged by mechanical stresses not only in the primary tumour but also during metastasis. However, this latter has seldom been stu...

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Autores principales: Fanfone, Deborah, Wu, Zhichong, Mammi, Jade, Berthenet, Kevin, Neves, David, Weber, Kathrin, Halaburkova, Andrea, Virard, François, Bunel, Félix, Jamard, Catherine, Hernandez-Vargas, Hector, Tait, Stephen WG, Hennino, Ana, Ichim, Gabriel
Formato: Online Artículo Texto
Lenguaje:English
Publicado: eLife Sciences Publications, Ltd 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8903834/
https://www.ncbi.nlm.nih.gov/pubmed/35256052
http://dx.doi.org/10.7554/eLife.73150
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author Fanfone, Deborah
Wu, Zhichong
Mammi, Jade
Berthenet, Kevin
Neves, David
Weber, Kathrin
Halaburkova, Andrea
Virard, François
Bunel, Félix
Jamard, Catherine
Hernandez-Vargas, Hector
Tait, Stephen WG
Hennino, Ana
Ichim, Gabriel
author_facet Fanfone, Deborah
Wu, Zhichong
Mammi, Jade
Berthenet, Kevin
Neves, David
Weber, Kathrin
Halaburkova, Andrea
Virard, François
Bunel, Félix
Jamard, Catherine
Hernandez-Vargas, Hector
Tait, Stephen WG
Hennino, Ana
Ichim, Gabriel
author_sort Fanfone, Deborah
collection PubMed
description Mechanical stress is known to fuel several hallmarks of cancer, ranging from genome instability to uncontrolled proliferation or invasion. Cancer cells are constantly challenged by mechanical stresses not only in the primary tumour but also during metastasis. However, this latter has seldom been studied with regards to mechanobiology, in particular resistance to anoikis, a cell death programme triggered by loss of cell adhesion. Here, we show in vitro that migrating breast cancer cells develop resistance to anoikis following their passage through microporous membranes mimicking confined migration (CM), a mechanical constriction that cancer cells encounter during metastasis. This CM-induced resistance was mediated by Inhibitory of Apoptosis Proteins, and sensitivity to anoikis could be restored after their inhibition using second mitochondria-derived activator of caspase (SMAC) mimetics. Anoikis-resistant mechanically stressed cancer cells displayed enhanced cell motility and evasion from natural killer cell-mediated immune surveillance, as well as a marked advantage to form lung metastatic lesions in mice. Our findings reveal that CM increases the metastatic potential of breast cancer cells.
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spelling pubmed-89038342022-03-09 Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness Fanfone, Deborah Wu, Zhichong Mammi, Jade Berthenet, Kevin Neves, David Weber, Kathrin Halaburkova, Andrea Virard, François Bunel, Félix Jamard, Catherine Hernandez-Vargas, Hector Tait, Stephen WG Hennino, Ana Ichim, Gabriel eLife Cancer Biology Mechanical stress is known to fuel several hallmarks of cancer, ranging from genome instability to uncontrolled proliferation or invasion. Cancer cells are constantly challenged by mechanical stresses not only in the primary tumour but also during metastasis. However, this latter has seldom been studied with regards to mechanobiology, in particular resistance to anoikis, a cell death programme triggered by loss of cell adhesion. Here, we show in vitro that migrating breast cancer cells develop resistance to anoikis following their passage through microporous membranes mimicking confined migration (CM), a mechanical constriction that cancer cells encounter during metastasis. This CM-induced resistance was mediated by Inhibitory of Apoptosis Proteins, and sensitivity to anoikis could be restored after their inhibition using second mitochondria-derived activator of caspase (SMAC) mimetics. Anoikis-resistant mechanically stressed cancer cells displayed enhanced cell motility and evasion from natural killer cell-mediated immune surveillance, as well as a marked advantage to form lung metastatic lesions in mice. Our findings reveal that CM increases the metastatic potential of breast cancer cells. eLife Sciences Publications, Ltd 2022-03-08 /pmc/articles/PMC8903834/ /pubmed/35256052 http://dx.doi.org/10.7554/eLife.73150 Text en © 2022, Fanfone et al https://creativecommons.org/licenses/by/4.0/This article is distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/) , which permits unrestricted use and redistribution provided that the original author and source are credited.
spellingShingle Cancer Biology
Fanfone, Deborah
Wu, Zhichong
Mammi, Jade
Berthenet, Kevin
Neves, David
Weber, Kathrin
Halaburkova, Andrea
Virard, François
Bunel, Félix
Jamard, Catherine
Hernandez-Vargas, Hector
Tait, Stephen WG
Hennino, Ana
Ichim, Gabriel
Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
title Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
title_full Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
title_fullStr Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
title_full_unstemmed Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
title_short Confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
title_sort confined migration promotes cancer metastasis through resistance to anoikis and increased invasiveness
topic Cancer Biology
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8903834/
https://www.ncbi.nlm.nih.gov/pubmed/35256052
http://dx.doi.org/10.7554/eLife.73150
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