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Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression
| Autores principales: | , , , , , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Nature Publishing Group UK
2022
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8904824/ https://www.ncbi.nlm.nih.gov/pubmed/35260598 http://dx.doi.org/10.1038/s41467-022-28923-3 |
| _version_ | 1784665031486472192 |
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| author | Dogliotti, Giada Kullmann, Lars Dhumale, Pratibha Thiele, Christian Panichkina, Olga Mendl, Gudrun Houben, Roland Haferkamp, Sebastian Püschel, Andreas W. Krahn, Michael P. |
| author_facet | Dogliotti, Giada Kullmann, Lars Dhumale, Pratibha Thiele, Christian Panichkina, Olga Mendl, Gudrun Houben, Roland Haferkamp, Sebastian Püschel, Andreas W. Krahn, Michael P. |
| author_sort | Dogliotti, Giada |
| collection | PubMed |
| description | |
| format | Online Article Text |
| id | pubmed-8904824 |
| institution | National Center for Biotechnology Information |
| language | English |
| publishDate | 2022 |
| publisher | Nature Publishing Group UK |
| record_format | MEDLINE/PubMed |
| spelling | pubmed-89048242022-03-23 Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression Dogliotti, Giada Kullmann, Lars Dhumale, Pratibha Thiele, Christian Panichkina, Olga Mendl, Gudrun Houben, Roland Haferkamp, Sebastian Püschel, Andreas W. Krahn, Michael P. Nat Commun Author Correction Nature Publishing Group UK 2022-03-08 /pmc/articles/PMC8904824/ /pubmed/35260598 http://dx.doi.org/10.1038/s41467-022-28923-3 Text en © The Author(s) 2022 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
| spellingShingle | Author Correction Dogliotti, Giada Kullmann, Lars Dhumale, Pratibha Thiele, Christian Panichkina, Olga Mendl, Gudrun Houben, Roland Haferkamp, Sebastian Püschel, Andreas W. Krahn, Michael P. Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression |
| title | Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression |
| title_full | Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression |
| title_fullStr | Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression |
| title_full_unstemmed | Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression |
| title_short | Author Correction: Membrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression |
| title_sort | author correction: membrane-binding and activation of lkb1 by phosphatidic acid is essential for development and tumour suppression |
| topic | Author Correction |
| url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8904824/ https://www.ncbi.nlm.nih.gov/pubmed/35260598 http://dx.doi.org/10.1038/s41467-022-28923-3 |
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