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A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application
Alpha-1 antitrypsin (AAT) is the canonical serine protease inhibitor of neutrophil-derived proteases and can modulate innate immune mechanisms through its anti-inflammatory activities mediated by a broad spectrum of protein, cytokine, and cell surface interactions. AAT contains a reactive methionine...
Autores principales: | , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8910375/ https://www.ncbi.nlm.nih.gov/pubmed/35269582 http://dx.doi.org/10.3390/ijms23052441 |
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author | O’Brien, Michael E. Murray, Grace Gogoi, Debananda Yusuf, Azeez McCarthy, Cormac Wormald, Mark R. Casey, Michelle Gabillard-Lefort, Claudie McElvaney, Noel G. Reeves, Emer P. |
author_facet | O’Brien, Michael E. Murray, Grace Gogoi, Debananda Yusuf, Azeez McCarthy, Cormac Wormald, Mark R. Casey, Michelle Gabillard-Lefort, Claudie McElvaney, Noel G. Reeves, Emer P. |
author_sort | O’Brien, Michael E. |
collection | PubMed |
description | Alpha-1 antitrypsin (AAT) is the canonical serine protease inhibitor of neutrophil-derived proteases and can modulate innate immune mechanisms through its anti-inflammatory activities mediated by a broad spectrum of protein, cytokine, and cell surface interactions. AAT contains a reactive methionine residue that is critical for its protease-specific binding capacity, whereby AAT entraps the protease on cleavage of its reactive centre loop, neutralises its activity by key changes in its tertiary structure, and permits removal of the AAT-protease complex from the circulation. Recently, however, the immunomodulatory role of AAT has come increasingly to the fore with several prominent studies focused on lipid or protein-protein interactions that are predominantly mediated through electrostatic, glycan, or hydrophobic potential binding sites. The aim of this review was to investigate the spectrum of AAT molecular interactions, with newer studies supporting a potential therapeutic paradigm for AAT augmentation therapy in disorders in which a chronic immune response is strongly linked. |
format | Online Article Text |
id | pubmed-8910375 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | MDPI |
record_format | MEDLINE/PubMed |
spelling | pubmed-89103752022-03-11 A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application O’Brien, Michael E. Murray, Grace Gogoi, Debananda Yusuf, Azeez McCarthy, Cormac Wormald, Mark R. Casey, Michelle Gabillard-Lefort, Claudie McElvaney, Noel G. Reeves, Emer P. Int J Mol Sci Review Alpha-1 antitrypsin (AAT) is the canonical serine protease inhibitor of neutrophil-derived proteases and can modulate innate immune mechanisms through its anti-inflammatory activities mediated by a broad spectrum of protein, cytokine, and cell surface interactions. AAT contains a reactive methionine residue that is critical for its protease-specific binding capacity, whereby AAT entraps the protease on cleavage of its reactive centre loop, neutralises its activity by key changes in its tertiary structure, and permits removal of the AAT-protease complex from the circulation. Recently, however, the immunomodulatory role of AAT has come increasingly to the fore with several prominent studies focused on lipid or protein-protein interactions that are predominantly mediated through electrostatic, glycan, or hydrophobic potential binding sites. The aim of this review was to investigate the spectrum of AAT molecular interactions, with newer studies supporting a potential therapeutic paradigm for AAT augmentation therapy in disorders in which a chronic immune response is strongly linked. MDPI 2022-02-23 /pmc/articles/PMC8910375/ /pubmed/35269582 http://dx.doi.org/10.3390/ijms23052441 Text en © 2022 by the authors. https://creativecommons.org/licenses/by/4.0/Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). |
spellingShingle | Review O’Brien, Michael E. Murray, Grace Gogoi, Debananda Yusuf, Azeez McCarthy, Cormac Wormald, Mark R. Casey, Michelle Gabillard-Lefort, Claudie McElvaney, Noel G. Reeves, Emer P. A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application |
title | A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application |
title_full | A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application |
title_fullStr | A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application |
title_full_unstemmed | A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application |
title_short | A Review of Alpha-1 Antitrypsin Binding Partners for Immune Regulation and Potential Therapeutic Application |
title_sort | review of alpha-1 antitrypsin binding partners for immune regulation and potential therapeutic application |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8910375/ https://www.ncbi.nlm.nih.gov/pubmed/35269582 http://dx.doi.org/10.3390/ijms23052441 |
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