Cargando…
Mitochondrial Protein Akap1 Deletion Exacerbates Endoplasmic Reticulum Stress in Mice Exposed to Hyperoxia
Acute lung injury (ALI) and its severe manifestation, acute respiratory distress syndrome (ARDS), are treated with high concentrations of supplementary oxygen. However, prolonged exposure to high oxygen concentrations stimulates the production of reactive oxygen species (ROS), which damages the mito...
Autores principales: | Sidramagowda Patil, Sahebgowda, Soundararajan, Ramani, Fukumoto, Jutaro, Breitzig, Mason, Hernández-Cuervo, Helena, Alleyn, Matthew, Lin, Muling, Narala, Venkata Ramireddy, Lockey, Richard, Kolliputi, Narasaiah, Galam, Lakshmi |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8964370/ https://www.ncbi.nlm.nih.gov/pubmed/35370705 http://dx.doi.org/10.3389/fphar.2022.762840 |
Ejemplares similares
-
Alda-1 Attenuates Hyperoxia-Induced Acute Lung Injury in Mice
por: Sidramagowda Patil, Sahebgowda, et al.
Publicado: (2021) -
Alda-1 attenuates hyperoxia-induced mitochondrial dysfunction in lung vascular endothelial cells
por: Patil, Sahebgowda Sidramagowda, et al.
Publicado: (2019) -
A-Kinase Anchor Protein 1 deficiency causes mitochondrial dysfunction in mouse model of hyperoxia induced acute lung injury
por: Soundararajan, Ramani, et al.
Publicado: (2022) -
BMI1 Silencing Induces Mitochondrial Dysfunction in Lung Epithelial Cells Exposed to Hyperoxia
por: Hernández-Cuervo, Helena, et al.
Publicado: (2022) -
Aberrant Expression of ACO1 in Vasculatures Parallels Progression of Idiopathic Pulmonary Fibrosis
por: Fukumoto, Jutaro, et al.
Publicado: (2022)