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Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury
Mitochondria release many damage-associated molecular patterns (DAMPs) when cells are damaged or stressed, with mitochondrial DNA (mtDNA) being. MtDNA activates innate immune responses and induces inflammation through the TLR-9, NLRP3 inflammasome, and cGAS-STING signaling pathways. Released inflamm...
Autores principales: | , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8966724/ https://www.ncbi.nlm.nih.gov/pubmed/35370747 http://dx.doi.org/10.3389/fphar.2022.854994 |
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author | Liao, Shishi Luo, Jie Kadier, Tulanisa Ding, Ke Chen, Rong Meng, Qingtao |
author_facet | Liao, Shishi Luo, Jie Kadier, Tulanisa Ding, Ke Chen, Rong Meng, Qingtao |
author_sort | Liao, Shishi |
collection | PubMed |
description | Mitochondria release many damage-associated molecular patterns (DAMPs) when cells are damaged or stressed, with mitochondrial DNA (mtDNA) being. MtDNA activates innate immune responses and induces inflammation through the TLR-9, NLRP3 inflammasome, and cGAS-STING signaling pathways. Released inflammatory factors cause damage to intestinal barrier function. Many bacteria and endotoxins migrate to the circulatory system and lymphatic system, leading to systemic inflammatory response syndrome (SIRS) and even damaging the function of multiple organs throughout the body. This process may ultimately lead to multiple organ dysfunction syndrome (MODS). Recent studies have shown that various factors, such as the release of mtDNA and the massive infiltration of inflammatory factors, can cause intestinal ischemia/reperfusion (I/R) injury. This destroys intestinal barrier function, induces an inflammatory storm, leads to SIRS, increases the vulnerability of organs, and develops into MODS. Mitophagy eliminates dysfunctional mitochondria to maintain cellular homeostasis. This review discusses mtDNA release during the pathogenesis of intestinal I/R and summarizes methods for the prevention or treatment of intestinal I/R. We also discuss the effects of inflammation and increased intestinal barrier permeability on drugs. |
format | Online Article Text |
id | pubmed-8966724 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | Frontiers Media S.A. |
record_format | MEDLINE/PubMed |
spelling | pubmed-89667242022-03-31 Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury Liao, Shishi Luo, Jie Kadier, Tulanisa Ding, Ke Chen, Rong Meng, Qingtao Front Pharmacol Pharmacology Mitochondria release many damage-associated molecular patterns (DAMPs) when cells are damaged or stressed, with mitochondrial DNA (mtDNA) being. MtDNA activates innate immune responses and induces inflammation through the TLR-9, NLRP3 inflammasome, and cGAS-STING signaling pathways. Released inflammatory factors cause damage to intestinal barrier function. Many bacteria and endotoxins migrate to the circulatory system and lymphatic system, leading to systemic inflammatory response syndrome (SIRS) and even damaging the function of multiple organs throughout the body. This process may ultimately lead to multiple organ dysfunction syndrome (MODS). Recent studies have shown that various factors, such as the release of mtDNA and the massive infiltration of inflammatory factors, can cause intestinal ischemia/reperfusion (I/R) injury. This destroys intestinal barrier function, induces an inflammatory storm, leads to SIRS, increases the vulnerability of organs, and develops into MODS. Mitophagy eliminates dysfunctional mitochondria to maintain cellular homeostasis. This review discusses mtDNA release during the pathogenesis of intestinal I/R and summarizes methods for the prevention or treatment of intestinal I/R. We also discuss the effects of inflammation and increased intestinal barrier permeability on drugs. Frontiers Media S.A. 2022-03-16 /pmc/articles/PMC8966724/ /pubmed/35370747 http://dx.doi.org/10.3389/fphar.2022.854994 Text en Copyright © 2022 Liao, Luo, Kadier, Ding, Chen and Meng. https://creativecommons.org/licenses/by/4.0/This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms. |
spellingShingle | Pharmacology Liao, Shishi Luo, Jie Kadier, Tulanisa Ding, Ke Chen, Rong Meng, Qingtao Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury |
title | Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury |
title_full | Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury |
title_fullStr | Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury |
title_full_unstemmed | Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury |
title_short | Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury |
title_sort | mitochondrial dna release contributes to intestinal ischemia/reperfusion injury |
topic | Pharmacology |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8966724/ https://www.ncbi.nlm.nih.gov/pubmed/35370747 http://dx.doi.org/10.3389/fphar.2022.854994 |
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