Cargando…
Beta-Amyloid (Aβ(1-42)) Increases the Expression of NKCC1 in the Mouse Hippocampus
Alzheimer’s disease (AD) is a neurodegenerative disorder with an increasing need for developing disease-modifying treatments as current therapies only provide marginal symptomatic relief. Recent evidence suggests the γ-aminobutyric acid (GABA) neurotransmitter system undergoes remodeling in AD, disr...
Autores principales: | Lam, Patricia, Vinnakota, Chitra, Guzmán, Beatriz Calvo-Flores, Newland, Julia, Peppercorn, Katie, Tate, Warren P., Waldvogel, Henry J., Faull, Richard L. M., Kwakowsky, Andrea |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9027496/ https://www.ncbi.nlm.nih.gov/pubmed/35458638 http://dx.doi.org/10.3390/molecules27082440 |
Ejemplares similares
-
The Acute Effects of Amyloid-Beta(1–42) on Glutamatergic Receptor and Transporter Expression in the Mouse Hippocampus
por: Yeung, Jason H. Y., et al.
Publicado: (2020) -
Amyloid-Beta(1-42) -Induced Increase in GABAergic Tonic Conductance in Mouse Hippocampal CA1 Pyramidal Cells
por: Calvo-Flores Guzmán, Beatriz, et al.
Publicado: (2020) -
An α5 GABAA Receptor Inverse Agonist, α5IA, Attenuates Amyloid Beta-Induced Neuronal Death in Mouse Hippocampal Cultures
por: Vinnakota, Chitra, et al.
Publicado: (2020) -
The Interplay Between Beta-Amyloid 1–42 (Aβ(1–42))-Induced Hippocampal Inflammatory Response, p-tau, Vascular Pathology, and Their Synergistic Contributions to Neuronal Death and Behavioral Deficits
por: Calvo-Flores Guzmán, Beatriz, et al.
Publicado: (2020) -
Treatment of beta amyloid 1–42 (Aβ(1–42))-induced basal forebrain cholinergic damage by a non-classical estrogen signaling activator in vivo
por: Kwakowsky, Andrea, et al.
Publicado: (2016)