Cargando…

Control of CRK-RAC1 activity by the miR-1/206/133 miRNA family is essential for neuromuscular junction function

Formation and maintenance of neuromuscular junctions (NMJs) are essential for skeletal muscle function, allowing voluntary movements and maintenance of the muscle tone, thereby preventing atrophy. Generation of NMJs depends on the interaction of motor neurons with skeletal muscle fibers, which initi...

Descripción completa

Detalles Bibliográficos
Autores principales:	Klockner, Ina, Schutt, Christian, Gerhardt, Theresa, Boettger, Thomas, Braun, Thomas
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Nature Publishing Group UK 2022
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9178026/ https://www.ncbi.nlm.nih.gov/pubmed/35676269 http://dx.doi.org/10.1038/s41467-022-30778-7

Ejemplares similares

The miR-206/133b cluster is dispensable for development, survival and regeneration of skeletal muscle
por: Boettger, Thomas, et al.
Publicado: (2014)

MiRNA-1/133a Clusters Regulate Adrenergic Control of Cardiac Repolarization
por: Besser, Johannes, et al.
Publicado: (2014)

FAIM Is Regulated by MiR-206, MiR-1-3p and MiR-133b
por: Coccia, Elena, et al.
Publicado: (2020)

Expression of miR-1, miR-133a, miR-133b and miR-206 increases during development of human skeletal muscle
por: Koutsoulidou, Andrie, et al.
Publicado: (2011)

Age-Related Exosomal and Endogenous Expression Patterns of miR-1, miR-133a, miR-133b, and miR-206 in Skeletal Muscles
por: Mytidou, Chrystalla, et al.
Publicado: (2021)

miR-1/133a Clusters Cooperatively Specify the Cardiomyogenic Lineage by Adjustment of Myocardin Levels during Embryonic Heart Development
por: Wystub, Katharina, et al.
Publicado: (2013)

Repression of Osmr and Fgfr1 by miR-1/133a prevents cardiomyocyte dedifferentiation and cell cycle entry in the adult heart
por: Valussi, Melissa, et al.
Publicado: (2021)

miR-206/133b Cluster: A Weapon against Lung Cancer?
por: Pan, Jing-Yu, et al.
Publicado: (2017)

Expression Patterns of Muscle-Specific miR-133b and miR-206 Correlate with Nutritional Status and Sarcopenia
por: Iannone, Francesca, et al.
Publicado: (2020)

Expression of miRNAs miR-133b and miR-206 in the Il17a/f Locus Is Co-Regulated with IL-17 Production in αβ and γδ T Cells
por: Haas, Jan D., et al.
Publicado: (2011)

Characterization and Functional Analysis of Extracellular Vesicles and Muscle-Abundant miRNAs (miR-1, miR-133a, and miR-206) in C(2)C(12) Myocytes and mdx Mice
por: Matsuzaka, Yasunari, et al.
Publicado: (2016)

Linc‐MYH configures INO80 to regulate muscle stem cell numbers and skeletal muscle hypertrophy
por: Schutt, Christian, et al.
Publicado: (2020)

Expression of miRNA-206 and miRNA-145 in breast cancer and correlation with prognosis
por: Quan, Yan, et al.
Publicado: (2018)

Capability of the viral NS1 proteins to bind to the cellular adaptor proteins Crk and CrkL determines sensitivity of influenza viruses to Crk/CrkL expression
por: Hrincius, ER, et al.
Publicado: (2009)

Palmitic Acid Impairs Myogenesis and Alters Temporal Expression of miR-133a and miR-206 in C2C12 Myoblasts
por: da Paixão, Ailma O., et al.
Publicado: (2021)

Levels matter: miR-206 and cyclin D1
por: Crescenzi, Marco
Publicado: (2013)

Aberrant expressions of miRNA-206 target, FN1, in multifactorial Hirschsprung disease
por: Gunadi, et al.
Publicado: (2019)

miRNA 206 and miRNA 574-5p are highly expression in coronary artery disease
por: Zhou, Jianqing, et al.
Publicado: (2016)

Crk and CrkL as Therapeutic Targets for Cancer Treatment
por: Park, Taeju
Publicado: (2021)

Smad8 Is Increased in Duchenne Muscular Dystrophy and Suppresses miR-1, miR-133a, and miR-133b
por: Lopez, Michael A., et al.
Publicado: (2022)

microRNA-1/133a and microRNA-206/133b clusters: Dysregulation and functional roles in human cancers
por: Nohata, Nijiro, et al.
Publicado: (2012)

Decrease in the expression of muscle-specific miRNAs, miR-133a and miR-1, in myoblasts with replicative senescence
por: Shintani-Ishida, Kaori, et al.
Publicado: (2023)

A heterozygous variant in the human cardiac miR-133 gene, MIR133A2, alters miRNA duplex processing and strand abundance
por: Ohanian, Monique, et al.
Publicado: (2013)

MiR-206 improves intervertebral disk degeneration by targeting GJA1
por: Zhou, Peng, et al.
Publicado: (2022)

MYOD1 inhibits avian adipocyte differentiation via miRNA-206/KLF4 axis
por: Wang, Zheng, et al.
Publicado: (2021)

Role of miR-1 and miR-133a in myocardial ischemic postconditioning
por: He, Bin, et al.
Publicado: (2011)

Crk and CrkL adaptor proteins: networks for physiological and pathological signaling
por: Birge, Raymond B, et al.
Publicado: (2009)

Serum miRNAs miR-23a, 206, and 499 as Potential Biomarkers for Skeletal Muscle Atrophy
por: Wang, Fei, et al.
Publicado: (2017)

uPAR promotes formation of the p130Cas–Crk complex to activate Rac through DOCK180
por: Smith, Harvey W., et al.
Publicado: (2008)

LINC00888 promoted tumorigenicity of melanoma via miR-126/CRK signaling axis
por: Lu, Wei, et al.
Publicado: (2018)

Expression of miRNA1, miRNA133, miRNA191, and miRNA24, as Good Biomarkers, in Non-Small Cell Lung Cancer Using Real-Time PCR Method
por: Kazempour Dizaji, Mehdi, et al.
Publicado: (2022)

Crk gets hijacked
por: Van Epps, Heather L.
Publicado: (2005)

Cracking Cancer with CRK
por: Uthamacumaran, Abicumaran, et al.
Publicado: (2009)

Combination of miRNA499 and miRNA133 Exerts a Synergic Effect on Cardiac Differentiation
por: Pisano, Federica, et al.
Publicado: (2015)

The miRNA Pathway Controls Rapid Changes in Activity-Dependent Synaptic Structure at the Drosophila melanogaster Neuromuscular Junction
por: Nesler, Katherine R., et al.
Publicado: (2013)

Purification of pseudopodia from polarized cells reveals redistribution and activation of Rac through assembly of a CAS/Crk scaffold
por: Cho, Samuel Y., et al.
Publicado: (2002)

Responsible Genes for Neuronal Migration in the Chromosome 17p13.3: Beyond Pafah1b1(Lis1), Crk and Ywhae(14-3-3ε)
por: Liu, Xiaonan, et al.
Publicado: (2021)

Crk1/2 and CrkL form a hetero-oligomer and functionally complement each other during podocyte morphogenesis
por: George, Britta, et al.
Publicado: (2014)

MyoD inhibits Fstl1 and Utrn expression by inducing transcription of miR-206
por: Rosenberg, Miriam I., et al.
Publicado: (2006)

Selective repression of the oncogene cyclin D1 by the tumor suppressor miR-206 in cancers
por: Elliman, S J, et al.
Publicado: (2014)

Cannot write session to /tmp/vufind_sessions/sess_mi05qicli282sj3s7a1jpcde2n