Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link

To date the true global incidence of intrahepatic cholangiocarcinoma (iCCA) and the underlying risk factors remain to be fully defined, in particular, the role of occupational and environmental factors. Currently, the putative role of asbestos exposure as a risk factor for iCCA is gaining increased...

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Detalles Bibliográficos
Autores principales: Brandi, Giovanni, Straif, Kurt, Mandrioli, Daniele, Curti, Stefania, Mattioli, Stefano, Tavolari, Simona
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Ubiquity Press 2022
Materias:
Letters to the Editor
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9205375/
https://www.ncbi.nlm.nih.gov/pubmed/35755315
http://dx.doi.org/10.5334/aogh.3660
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author Brandi, Giovanni
Straif, Kurt
Mandrioli, Daniele
Curti, Stefania
Mattioli, Stefano
Tavolari, Simona
author_facet Brandi, Giovanni
Straif, Kurt
Mandrioli, Daniele
Curti, Stefania
Mattioli, Stefano
Tavolari, Simona
author_sort Brandi, Giovanni
collection PubMed
description To date the true global incidence of intrahepatic cholangiocarcinoma (iCCA) and the underlying risk factors remain to be fully defined, in particular, the role of occupational and environmental factors. Currently, the putative role of asbestos exposure as a risk factor for iCCA is gaining increased attention in the international scientific community and agencies. In this commentary we review and integrate available epidemiological and mechanistic evidences that support a potential role of asbestos exposure in iCCA etiology.
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spelling pubmed-92053752022-06-23 Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link Brandi, Giovanni Straif, Kurt Mandrioli, Daniele Curti, Stefania Mattioli, Stefano Tavolari, Simona Ann Glob Health Letters to the Editor To date the true global incidence of intrahepatic cholangiocarcinoma (iCCA) and the underlying risk factors remain to be fully defined, in particular, the role of occupational and environmental factors. Currently, the putative role of asbestos exposure as a risk factor for iCCA is gaining increased attention in the international scientific community and agencies. In this commentary we review and integrate available epidemiological and mechanistic evidences that support a potential role of asbestos exposure in iCCA etiology. Ubiquity Press 2022-06-13 /pmc/articles/PMC9205375/ /pubmed/35755315 http://dx.doi.org/10.5334/aogh.3660 Text en Copyright: © 2022 The Author(s) https://creativecommons.org/licenses/by/4.0/This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International License (CC-BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. See http://creativecommons.org/licenses/by/4.0/.
spellingShingle Letters to the Editor
Brandi, Giovanni
Straif, Kurt
Mandrioli, Daniele
Curti, Stefania
Mattioli, Stefano
Tavolari, Simona
Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link
title Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link
title_full Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link
title_fullStr Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link
title_full_unstemmed Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link
title_short Exposure to Asbestos and Increased Intrahepatic Cholangiocarcinoma Risk: Growing Evidences of a Putative Causal Link
title_sort exposure to asbestos and increased intrahepatic cholangiocarcinoma risk: growing evidences of a putative causal link
topic Letters to the Editor
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9205375/
https://www.ncbi.nlm.nih.gov/pubmed/35755315
http://dx.doi.org/10.5334/aogh.3660
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