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Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway
| Autores principales: | , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2022
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9248095/ https://www.ncbi.nlm.nih.gov/pubmed/35773717 http://dx.doi.org/10.1186/s13578-022-00837-z |
| _version_ | 1784739300054663168 |
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| author | Zhang, Junwei Yang, Suxia Chen, Fang Li, Huicong Chen, Baoping |
| author_facet | Zhang, Junwei Yang, Suxia Chen, Fang Li, Huicong Chen, Baoping |
| author_sort | Zhang, Junwei |
| collection | PubMed |
| description | |
| format | Online Article Text |
| id | pubmed-9248095 |
| institution | National Center for Biotechnology Information |
| language | English |
| publishDate | 2022 |
| publisher | BioMed Central |
| record_format | MEDLINE/PubMed |
| spelling | pubmed-92480952022-07-02 Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway Zhang, Junwei Yang, Suxia Chen, Fang Li, Huicong Chen, Baoping Cell Biosci Editorial Expression of Concern BioMed Central 2022-06-30 /pmc/articles/PMC9248095/ /pubmed/35773717 http://dx.doi.org/10.1186/s13578-022-00837-z Text en © The Author(s) 2022 https://creativecommons.org/licenses/by/4.0/Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/ (https://creativecommons.org/publicdomain/zero/1.0/) ) applies to the data made available in this article, unless otherwise stated in a credit line to the data. |
| spellingShingle | Editorial Expression of Concern Zhang, Junwei Yang, Suxia Chen, Fang Li, Huicong Chen, Baoping Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway |
| title | Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway |
| title_full | Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway |
| title_fullStr | Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway |
| title_full_unstemmed | Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway |
| title_short | Editorial Expression of Concern: Ginkgetin aglycone ameliorates LPS-induced acute kidney injury by activating SIRT1 via inhibiting the NF-κB signaling pathway |
| title_sort | editorial expression of concern: ginkgetin aglycone ameliorates lps-induced acute kidney injury by activating sirt1 via inhibiting the nf-κb signaling pathway |
| topic | Editorial Expression of Concern |
| url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9248095/ https://www.ncbi.nlm.nih.gov/pubmed/35773717 http://dx.doi.org/10.1186/s13578-022-00837-z |
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