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Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report

Guillain-Barre syndrome is an acute demyelinating polyneuropathy disease which is autoimmune in nature and usually follows gastrointestinal or respiratory infections. Dengue fever is however not a common trigger to the condition. Here, we report a patient who developed sensory predominant demyelinat...

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Detalles Bibliográficos
Autores principales: Payus, Alvin Oliver, Ibrahim, Azliza, Liew Sat Lin, Constance, Hui Jan, Tan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: S. Karger AG 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9251453/
https://www.ncbi.nlm.nih.gov/pubmed/35949203
http://dx.doi.org/10.1159/000524865
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author Payus, Alvin Oliver
Ibrahim, Azliza
Liew Sat Lin, Constance
Hui Jan, Tan
author_facet Payus, Alvin Oliver
Ibrahim, Azliza
Liew Sat Lin, Constance
Hui Jan, Tan
author_sort Payus, Alvin Oliver
collection PubMed
description Guillain-Barre syndrome is an acute demyelinating polyneuropathy disease which is autoimmune in nature and usually follows gastrointestinal or respiratory infections. Dengue fever is however not a common trigger to the condition. Here, we report a patient who developed sensory predominant demyelinating polyradiculopathy during febrile phase of dengue fever. It was later confirmed with serology test and nerve conduction study. He was successfully treated with intravenous immunoglobulin and discharged home well. The purpose of this case report is to highlight that Guillain-Barré syndrome can occur as an uncommon neurological complication of dengue fever which can occur during any phase of the illness.
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spelling pubmed-92514532022-08-09 Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report Payus, Alvin Oliver Ibrahim, Azliza Liew Sat Lin, Constance Hui Jan, Tan Case Rep Neurol Single Case − General Neurology Guillain-Barre syndrome is an acute demyelinating polyneuropathy disease which is autoimmune in nature and usually follows gastrointestinal or respiratory infections. Dengue fever is however not a common trigger to the condition. Here, we report a patient who developed sensory predominant demyelinating polyradiculopathy during febrile phase of dengue fever. It was later confirmed with serology test and nerve conduction study. He was successfully treated with intravenous immunoglobulin and discharged home well. The purpose of this case report is to highlight that Guillain-Barré syndrome can occur as an uncommon neurological complication of dengue fever which can occur during any phase of the illness. S. Karger AG 2022-06-17 /pmc/articles/PMC9251453/ /pubmed/35949203 http://dx.doi.org/10.1159/000524865 Text en Copyright © 2022 by The Author(s). Published by S. Karger AG, Basel https://creativecommons.org/licenses/by-nc/4.0/This article is licensed under the Creative Commons Attribution-NonCommercial-4.0 International License (CC BY-NC) (http://www.karger.com/Services/OpenAccessLicense). Usage and distribution for commercial purposes requires written permission.
spellingShingle Single Case − General Neurology
Payus, Alvin Oliver
Ibrahim, Azliza
Liew Sat Lin, Constance
Hui Jan, Tan
Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report
title Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report
title_full Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report
title_fullStr Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report
title_full_unstemmed Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report
title_short Sensory Predominant Guillain-Barré Syndrome Concomitant with Dengue Infection: A Case Report
title_sort sensory predominant guillain-barré syndrome concomitant with dengue infection: a case report
topic Single Case − General Neurology
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9251453/
https://www.ncbi.nlm.nih.gov/pubmed/35949203
http://dx.doi.org/10.1159/000524865
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