Adiponectin/AdiopR1 signaling prevents mitochondrial dysfunction and oxidative injury after traumatic brain injury in a SIRT3 dependent manner

Mitochondrial dysfunction and oxidative injury, which contribute to worsening of neurological deficits and poor clinical outcomes, are hallmarks of secondary brain injury after TBI. Adiponectin (APN), beyond its well-established regulatory effects on metabolism, is also essential for maintaining nor...

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Detalles Bibliográficos
Autores principales: Zhang, Shenghao, Wu, Xun, Wang, Jin, Shi, Yingwu, Hu, Qing, Cui, Wenxing, Bai, Hao, Zhou, Jinpeng, Du, Yong, Han, Liying, Li, Leiyang, Feng, Dayun, Ge, Shunnan, Qu, Yan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2022
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9287731/
https://www.ncbi.nlm.nih.gov/pubmed/35793583
http://dx.doi.org/10.1016/j.redox.2022.102390

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9287731/
https://www.ncbi.nlm.nih.gov/pubmed/35793583
http://dx.doi.org/10.1016/j.redox.2022.102390

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