p65/RelA NF‐κB fragments generated by RIPK3 activity regulate tumorigenicity, cell metabolism, and stemness characteristics

Receptor‐interacting protein kinase 3 (RIPK3) can induce necroptosis, apoptosis, or cell proliferation and is silenced in several hematological malignancies. We previously reported that RIPK3 activity independent of its kinase domain induces caspase‐mediated p65/RelA cleavage, resulting in N‐termina...

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Detalles Bibliográficos
Autores principales: Touil, Yasmine, Latreche‐Carton, Céline, Bouazzati, Hassiba El, Nugues, Anne‐Lucie, Jouy, Nathalie, Thuru, Xavier, Laine, William, Lepretre, Frederic, Figeac, Martin, Tardivel, Meryem, Kluza, Jérôme, Idziorek, Thierry, Quesnel, Bruno
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2021
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9299825/
https://www.ncbi.nlm.nih.gov/pubmed/34927768
http://dx.doi.org/10.1002/jcb.30198

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