YKL-40 changes are not detected in post-mortem brain of patients with Alzheimer’s disease and frontotemporal lobar degeneration

BACKGROUND: YKL-40 (Chitinase 3-like I) is increased in CSF of Alzheimer’s disease (AD) and frontotemporal lobar degeneration (FTLD) patients and is therefore considered a potential neuroinflammatory biomarker. Whether changed YKL-40 levels in the CSF reflect dysregulation of YKL-40 in the brain is...

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Detalles Bibliográficos
Autores principales: Hok-A-Hin, Yanaika S., Hoozemans, Jeroen J. M., Hu, William T., Wouters, Dorine, Howell, Jennifer C., Rábano, Alberto, van der Flier, Wiesje M., Pijnenburg, Yolande A. L., Teunissen, Charlotte E., del Campo, Marta
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2022
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9310415/
https://www.ncbi.nlm.nih.gov/pubmed/35879733
http://dx.doi.org/10.1186/s13195-022-01039-y

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