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Molecular Pathogenesis and Disease-modifying Therapies of Alzheimer’s Disease and Related Disorders
The deposition of amyloid β (Aβ) peptides as senile plaques and tau as neurofibrillary changes causes the hallmark neuropathological lesions of Alzheimer’s disease (AD), which are implicated in its pathogenesis and deemed as the prime target for disease-modifying therapies (DMTs). Aβ is produced by...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Japan Medical Association
2022
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9358130/ https://www.ncbi.nlm.nih.gov/pubmed/35992282 http://dx.doi.org/10.31662/jmaj.2022-0079 |