Cargando…
Oxidative Stress: Glutathione and Its Potential to Protect Methionine-35 of Aβ Peptide from Oxidation
[Image: see text] Alzheimer’s disease (AD) is the most common neurodegenerative disorder with heterogeneous etiology. Intracellular neurofibrillary tangles caused by tau (τ) protein phosphorylation and extracellular senile plaques caused by aggregation of amyloid-beta (Aβ) peptide are characteristic...
Autores principales: | Mandal, Pravat K., Roy, Rimil Guha, Samkaria, Avantika |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
American Chemical Society
2022
|
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9366984/ https://www.ncbi.nlm.nih.gov/pubmed/35967059 http://dx.doi.org/10.1021/acsomega.2c02760 |
Ejemplares similares
-
Oxidative Stress
Occurs Prior to Amyloid Aβ
Plaque Formation and Tau Phosphorylation in Alzheimer’s Disease:
Role of Glutathione and Metal Ions
por: Roy, Rimil Guha, et al.
Publicado: (2023) -
Effect of methionine-35 oxidation on the aggregation of amyloid-β peptide
por: Friedemann, Merlin, et al.
Publicado: (2015) -
Oxidation of Methionine-35 in Alzheimer’s amyloid-beta peptide and the aggregation of the oxidized peptide
por: Friedemann, Merlin, et al.
Publicado: (2015) -
Methionine-35 of Aβ(1–42): Importance for Oxidative Stress in Alzheimer Disease
por: Butterfield, D. Allan, et al.
Publicado: (2011) -
Blood Biomarkers
in Alzheimer’s Disease
por: Mandal, Pravat K., et al.
Publicado: (2023)