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Alcohol-related diseases and liver metastasis: Role of cell-free network communication

Alcohol intake is a risk factor for cancer development and metastatic disease progression. Extracellular vesicle (EV)-mediated interorgan communication is assumed to be significant in boosting tumorigenic pathways and disease progression. Recent research indicates that exosomes have a variety of rol...

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Detalles Bibliográficos
Autores principales: Muro, Manuel, Collados-Ros, Aurelia, Legaz, Isabel
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Baishideng Publishing Group Inc 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9403426/
https://www.ncbi.nlm.nih.gov/pubmed/36157111
http://dx.doi.org/10.3748/wjg.v28.i30.4231
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author Muro, Manuel
Collados-Ros, Aurelia
Legaz, Isabel
author_facet Muro, Manuel
Collados-Ros, Aurelia
Legaz, Isabel
author_sort Muro, Manuel
collection PubMed
description Alcohol intake is a risk factor for cancer development and metastatic disease progression. Extracellular vesicle (EV)-mediated interorgan communication is assumed to be significant in boosting tumorigenic pathways and disease progression. Recent research indicates that exosomes have a variety of roles in the development of cancer during pathophysiological conditions. The involvement of EV signaling during cancer progression in the alcohol environment is unknown. Therefore, understanding communication networks and the role of EVs as biomarkers can contribute significantly to developing strategies to address the serious public health problems associated with alcohol consumption and cancer.
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spelling pubmed-94034262022-09-23 Alcohol-related diseases and liver metastasis: Role of cell-free network communication Muro, Manuel Collados-Ros, Aurelia Legaz, Isabel World J Gastroenterol Letter to the Editor Alcohol intake is a risk factor for cancer development and metastatic disease progression. Extracellular vesicle (EV)-mediated interorgan communication is assumed to be significant in boosting tumorigenic pathways and disease progression. Recent research indicates that exosomes have a variety of roles in the development of cancer during pathophysiological conditions. The involvement of EV signaling during cancer progression in the alcohol environment is unknown. Therefore, understanding communication networks and the role of EVs as biomarkers can contribute significantly to developing strategies to address the serious public health problems associated with alcohol consumption and cancer. Baishideng Publishing Group Inc 2022-08-14 2022-08-14 /pmc/articles/PMC9403426/ /pubmed/36157111 http://dx.doi.org/10.3748/wjg.v28.i30.4231 Text en ©The Author(s) 2022. Published by Baishideng Publishing Group Inc. All rights reserved. https://creativecommons.org/licenses/by-nc/4.0/This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: https://creativecommons.org/Licenses/by-nc/4.0/
spellingShingle Letter to the Editor
Muro, Manuel
Collados-Ros, Aurelia
Legaz, Isabel
Alcohol-related diseases and liver metastasis: Role of cell-free network communication
title Alcohol-related diseases and liver metastasis: Role of cell-free network communication
title_full Alcohol-related diseases and liver metastasis: Role of cell-free network communication
title_fullStr Alcohol-related diseases and liver metastasis: Role of cell-free network communication
title_full_unstemmed Alcohol-related diseases and liver metastasis: Role of cell-free network communication
title_short Alcohol-related diseases and liver metastasis: Role of cell-free network communication
title_sort alcohol-related diseases and liver metastasis: role of cell-free network communication
topic Letter to the Editor
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9403426/
https://www.ncbi.nlm.nih.gov/pubmed/36157111
http://dx.doi.org/10.3748/wjg.v28.i30.4231
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