Cargando…
Notch Signaling Induced by Endoplasmic Reticulum Stress Regulates Cumulus-Oocyte Complex Expansion in Polycystic Ovary Syndrome
Endoplasmic reticulum (ER) stress activated in granulosa cells contributes to the pathophysiology of polycystic ovary syndrome (PCOS). In addition, recent studies have demonstrated that Notch signaling plays multiple roles in the ovary via cell-to-cell interactions. We hypothesized that ER stress ac...
Autores principales: | Koike, Hiroshi, Harada, Miyuki, Kusamoto, Akari, Kunitomi, Chisato, Xu, Zixin, Tanaka, Tsurugi, Urata, Yoko, Nose, Emi, Takahashi, Nozomi, Wada-Hiraike, Osamu, Hirota, Yasushi, Koga, Kaori, Osuga, Yutaka |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9405998/ https://www.ncbi.nlm.nih.gov/pubmed/36008931 http://dx.doi.org/10.3390/biom12081037 |
Ejemplares similares
-
Roles of endoplasmic reticulum stress in the pathophysiology of polycystic ovary syndrome
por: Koike, Hiroshi, et al.
Publicado: (2023) -
RF10 | PMON241 Temporal Changes in Phenotype and Gut microbiota in PCOS Mouse Model Induced by Prenatal Androgen Exposure.
por: Kusamoto, Akari, et al.
Publicado: (2022) -
MON-033 Androgen Increases the Accumulation of Advanced Glycation End Products in Granulosa Cells by Activating ER Stress in PCOS
por: Azhary, Jerilee Mariam Khong, et al.
Publicado: (2020) -
Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis
por: Takahashi, Nozomi, et al.
Publicado: (2017) -
Upregulation of Aryl Hydrocarbon Receptor in Granulosa Cells by Endoplasmic Reticulum Stress Contributes to the PCOS Pathophysiology
por: Kunitomi, Chisato, et al.
Publicado: (2021)