Cargando…
FAK Executes Anti-Senescence via Regulating EZH2 Signaling in Non-Small Cell Lung Cancer Cells
Focal adhesion kinase (FAK) is a non-receptor tyrosine kinase overexpressed in various cancer types that plays a critical role in tumor progression. Accumulating evidence suggests that targeting FAK, either alone or in combination with other agents, may serve as an effective therapeutic strategy for...
Autores principales: | Chuang, Hsiang-Hao, Huang, Ming-Shyan, Zhen, Yen-Yi, Chuang, Cheng-Hao, Lee, Ying-Ray, Hsiao, Michael, Yang, Chih-Jen |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9406208/ https://www.ncbi.nlm.nih.gov/pubmed/36009484 http://dx.doi.org/10.3390/biomedicines10081937 |
Ejemplares similares
-
Inhibition of FAK Signaling Elicits Lamin A/C-Associated Nuclear Deformity and Cellular Senescence
por: Chuang, Hsiang-Hao, et al.
Publicado: (2019) -
FAK in Cancer: From Mechanisms to Therapeutic Strategies
por: Chuang, Hsiang-Hao, et al.
Publicado: (2022) -
Targeting Pin1 for Modulation of Cell Motility and Cancer Therapy
por: Chuang, Hsiang-Hao, et al.
Publicado: (2021) -
Pin1 Is Involved in HDAC6-mediated Cancer Cell Motility
por: Chuang, Hsiang-Hao, et al.
Publicado: (2018) -
Pin1 coordinates HDAC6 upregulation with cell migration in lung cancer cells
por: Chuang, Hsiang-Hao, et al.
Publicado: (2020)