Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?

SARS-CoV-2 has caused devastating effects with over 550 million infections by July 2022 and approximately 6.4 million deaths [1]. Societal and economic impacts will reverberate for years, with continuous evolution of SARS-CoV-2 as it persistently spreads through the human population as exemplified b...

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Autores principales: Dockrell, David H., Russell, Clark D., McHugh, Brian, Fraser, Rupsha
Formato: Online Artículo Texto
Lenguaje:English
Publicado: European Respiratory Society 2022
Materias:
Editorial
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9411729/
https://www.ncbi.nlm.nih.gov/pubmed/36028257
http://dx.doi.org/10.1183/13993003.01521-2022
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author Dockrell, David H.
Russell, Clark D.
McHugh, Brian
Fraser, Rupsha
author_facet Dockrell, David H.
Russell, Clark D.
McHugh, Brian
Fraser, Rupsha
author_sort Dockrell, David H.
collection PubMed
description SARS-CoV-2 has caused devastating effects with over 550 million infections by July 2022 and approximately 6.4 million deaths [1]. Societal and economic impacts will reverberate for years, with continuous evolution of SARS-CoV-2 as it persistently spreads through the human population as exemplified by reduced activity of vaccines and monoclonals against Omicron BA.4 or BA.5 subvariants [2]. A greater understanding of pathogenesis and more tailored therapeutic approaches are therefore essential.
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spelling pubmed-94117292022-08-26 Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19? Dockrell, David H. Russell, Clark D. McHugh, Brian Fraser, Rupsha Eur Respir J Editorial SARS-CoV-2 has caused devastating effects with over 550 million infections by July 2022 and approximately 6.4 million deaths [1]. Societal and economic impacts will reverberate for years, with continuous evolution of SARS-CoV-2 as it persistently spreads through the human population as exemplified by reduced activity of vaccines and monoclonals against Omicron BA.4 or BA.5 subvariants [2]. A greater understanding of pathogenesis and more tailored therapeutic approaches are therefore essential. European Respiratory Society 2022-08-26 /pmc/articles/PMC9411729/ /pubmed/36028257 http://dx.doi.org/10.1183/13993003.01521-2022 Text en Copyright ©The authors 2022 https://creativecommons.org/licenses/by-nc/4.0/This version is distributed under the terms of the Creative Commons Attribution Non-Commercial Licence 4.0. For commercial reproduction rights and permissions contact permissions@ersnet.org (mailto:permissions@ersnet.org)
spellingShingle Editorial
Dockrell, David H.
Russell, Clark D.
McHugh, Brian
Fraser, Rupsha
Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?
title Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?
title_full Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?
title_fullStr Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?
title_full_unstemmed Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?
title_short Does autonomous macrophage-driven inflammation promote alveolar damage in COVID-19?
title_sort does autonomous macrophage-driven inflammation promote alveolar damage in covid-19?
topic Editorial
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9411729/
https://www.ncbi.nlm.nih.gov/pubmed/36028257
http://dx.doi.org/10.1183/13993003.01521-2022
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